| pubmed-article:3985118 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3985118 | lifeskim:mentions | umls-concept:C1135183 | lld:lifeskim |
| pubmed-article:3985118 | lifeskim:mentions | umls-concept:C1186983 | lld:lifeskim |
| pubmed-article:3985118 | lifeskim:mentions | umls-concept:C0333582 | lld:lifeskim |
| pubmed-article:3985118 | lifeskim:mentions | umls-concept:C0231174 | lld:lifeskim |
| pubmed-article:3985118 | lifeskim:mentions | umls-concept:C1710236 | lld:lifeskim |
| pubmed-article:3985118 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:3985118 | pubmed:dateCreated | 1985-5-10 | lld:pubmed |
| pubmed-article:3985118 | pubmed:abstractText | Calcific degeneration is the main cause of porcine bioprosthetic valve failure. This dystrophic phenomenon has been studied by transmission electron microscopy in 26 explants; six normally processed unimplanted xenografts and a pig aortic valve from the slaughterhouse served as controls. Loss of endothelial lining and proteoglycans were a regular finding in all the commercially processed valves. In order to detect initial calcifications, we investigated in particular areas apparently devoid of mineralization at x-ray. Three main ultramicroscopic features were found: 1) intracytoplasmic and interstitial calcospherulae in 22 explants, 2) calcified collagen fibrils in 15, and 3) platelike calcium deposits upon amorphous material in 3. X-ray diffraction and energy-dispersive microanalysis identified Ca2+ deposits as crystals of hydroxyapatite. From these findings there is evidence that debris and membrane fragments of the pig cusp cells represent one of the initial nuclei of calcification. | lld:pubmed |
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| pubmed-article:3985118 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3985118 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3985118 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:3985118 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3985118 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:3985118 | pubmed:issn | 0002-9440 | lld:pubmed |
| pubmed-article:3985118 | pubmed:author | pubmed-author:ValentaAA | lld:pubmed |
| pubmed-article:3985118 | pubmed:author | pubmed-author:ThieneGG | lld:pubmed |
| pubmed-article:3985118 | pubmed:author | pubmed-author:BortolottiUU | lld:pubmed |
| pubmed-article:3985118 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3985118 | pubmed:volume | 119 | lld:pubmed |
| pubmed-article:3985118 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3985118 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3985118 | pubmed:pagination | 12-21 | lld:pubmed |
| pubmed-article:3985118 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3985118 | pubmed:meshHeading | pubmed-meshheading:3985118-... | lld:pubmed |
| pubmed-article:3985118 | pubmed:year | 1985 | lld:pubmed |
| pubmed-article:3985118 | pubmed:articleTitle | Ultrastructural substrates of dystrophic calcification in porcine bioprosthetic valve failure. | lld:pubmed |
| pubmed-article:3985118 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3985118 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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