| pubmed-article:3856237 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3856237 | lifeskim:mentions | umls-concept:C0027260 | lld:lifeskim |
| pubmed-article:3856237 | lifeskim:mentions | umls-concept:C0023467 | lld:lifeskim |
| pubmed-article:3856237 | lifeskim:mentions | umls-concept:C0544886 | lld:lifeskim |
| pubmed-article:3856237 | lifeskim:mentions | umls-concept:C0015295 | lld:lifeskim |
| pubmed-article:3856237 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:3856237 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:3856237 | pubmed:dateCreated | 1985-3-27 | lld:pubmed |
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| pubmed-article:3856237 | pubmed:abstractText | A transforming N-ras gene has been cloned from acute myeloblastic leukemia bone marrow cells, in parallel with the N-ras gene derived from fibroblasts of the same patient. N-ras derived from fibroblasts lacked focus-forming activity in NIH/3T3 cells, indicating that gene activation in the leukemia cells must have occurred by a somatic event. Construction of chimeric molecules between the transforming and the normal N-ras genes and subsequent biological and sequence analysis of these constructs revealed that the transforming gene was altered by a point mutation changing amino acid 12 of the N-ras protein from glycine to aspartic acid. | lld:pubmed |
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| pubmed-article:3856237 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3856237 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3856237 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:3856237 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3856237 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:3856237 | pubmed:issn | 0027-8424 | lld:pubmed |
| pubmed-article:3856237 | pubmed:author | pubmed-author:MoroniCC | lld:pubmed |
| pubmed-article:3856237 | pubmed:author | pubmed-author:HallAA | lld:pubmed |
| pubmed-article:3856237 | pubmed:author | pubmed-author:GambkeCC | lld:pubmed |
| pubmed-article:3856237 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3856237 | pubmed:volume | 82 | lld:pubmed |
| pubmed-article:3856237 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3856237 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3856237 | pubmed:pagination | 879-82 | lld:pubmed |
| pubmed-article:3856237 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3856237 | pubmed:year | 1985 | lld:pubmed |
| pubmed-article:3856237 | pubmed:articleTitle | Activation of an N-ras gene in acute myeloblastic leukemia through somatic mutation in the first exon. | lld:pubmed |
| pubmed-article:3856237 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3856237 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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