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| pubmed-article:3597776 | lifeskim:mentions | umls-concept:C0034721 | lld:lifeskim |
| pubmed-article:3597776 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:3597776 | lifeskim:mentions | umls-concept:C0022677 | lld:lifeskim |
| pubmed-article:3597776 | lifeskim:mentions | umls-concept:C0003009 | lld:lifeskim |
| pubmed-article:3597776 | lifeskim:mentions | umls-concept:C1522821 | lld:lifeskim |
| pubmed-article:3597776 | lifeskim:mentions | umls-concept:C1704735 | lld:lifeskim |
| pubmed-article:3597776 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:3597776 | pubmed:dateCreated | 1987-8-4 | lld:pubmed |
| pubmed-article:3597776 | pubmed:abstractText | The early proximal convoluted tubule (PCT) is the site of 50% of bicarbonate reabsorption in the nephron, but its control by angiotensin II has not been previously studied. In vivo microperfusion was used in both the early and late PCT in Munich-Wistar rats. Systemic angiotensin II administration (20 ng/kg X min) or inhibition of endogenous angiotensin II activity with saralasin (1 microgram/kg X min) caused profound changes in bicarbonate absorption in the early PCT (169 +/- 25 and -187 +/- 15 peq/mm X min, respectively). Because the bicarbonate absorptive capacity of the early PCT under free-flow conditions is 500 peq/mm X min, angiotensin II administration or inhibition affected greater than 60% of proton secretion in this segment. Both agents less markedly affected bicarbonate absorption in the late PCT (+/- 28 peq/mm X min) or chloride absorption (+/- 68-99 peq/mm X min) in both the early and late PCT. Because of its potential for controlling the majority of bicarbonate absorption in the early PCT (hence greater than or equal to 30% of bicarbonate absorption in the entire nephron), angiotensin II may be a powerful physiologic regulator of renal acidification. | lld:pubmed |
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| pubmed-article:3597776 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:3597776 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:3597776 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3597776 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:3597776 | pubmed:issn | 0021-9738 | lld:pubmed |
| pubmed-article:3597776 | pubmed:author | pubmed-author:CoganM GMG | lld:pubmed |
| pubmed-article:3597776 | pubmed:author | pubmed-author:LiuF YFY | lld:pubmed |
| pubmed-article:3597776 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3597776 | pubmed:volume | 80 | lld:pubmed |
| pubmed-article:3597776 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3597776 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3597776 | pubmed:pagination | 272-5 | lld:pubmed |
| pubmed-article:3597776 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3597776 | pubmed:year | 1987 | lld:pubmed |
| pubmed-article:3597776 | pubmed:articleTitle | Angiotensin II: a potent regulator of acidification in the rat early proximal convoluted tubule. | lld:pubmed |
| pubmed-article:3597776 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3597776 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:3597776 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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