Linked Life Data

3213837

Source:http://linkedlifedata.com/resource/pubmed/id/3213837

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
6
pubmed:dateCreated
1989-2-23
pubmed:abstractText
This study was designed to investigate the hemodynamic effects of smoking in patients with congestive heart failure. Hemodynamic measurements were made in seven patients with heart failure before, during, and after smoking two unfiltered cigarettes (approximately 2.4 mg of nicotine). Smoking significantly (p less than .05) increased heart rate and decreased stroke volume. Cardiac output remained unchanged. Significant increases were noted in systemic systolic and diastolic blood pressures concomitant with significant increases in total systemic vascular resistance and systemic arteriolar resistance. Pulmonary arterial pressure and total pulmonary vascular resistance increased as well. Mean right atrial pressure and pulmonary capillary wedge pressure rose modestly during smoking. Finally, the double product, an index of myocardial oxygen consumption, increased considerably during the smoking and postsmoking periods. Smoking elicits significant hemodynamic effects in patients with congestive heart failure, many unfavorable and potentially detrimental.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:status
MEDLINE
pubmed:month
Dec
pubmed:issn
0002-9629
pubmed:author
pubmed:issnType
Print
pubmed:volume
296
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
377-80
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed:year
1988
pubmed:articleTitle
Hemodynamic effects of smoking in congestive heart failure.
pubmed:affiliation
Division of Cardiology, Ohio State University College of Medicine, Columbus 43210.
pubmed:publicationType
Journal Article