| pubmed-article:3126396 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3126396 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:3126396 | lifeskim:mentions | umls-concept:C0024518 | lld:lifeskim |
| pubmed-article:3126396 | lifeskim:mentions | umls-concept:C0205369 | lld:lifeskim |
| pubmed-article:3126396 | lifeskim:mentions | umls-concept:C1514468 | lld:lifeskim |
| pubmed-article:3126396 | pubmed:issue | 6159 | lld:pubmed |
| pubmed-article:3126396 | pubmed:dateCreated | 1988-4-8 | lld:pubmed |
| pubmed-article:3126396 | pubmed:abstractText | In mice the product of the Mlsa locus is an unusual antigen capable of interaction with certain products of the major histocompatibility locus (MHC) to form a ligand for a large portion of the T-cell alpha/beta receptor repertoire, including nearly all receptors that use V beta 8.1. The presence of Mlsa/MHC during T-cell development results in the deletion of T cells that express V beta 8.1, documenting the importance of clonal deletion in establishing tolerance to self antigens. | lld:pubmed |
| pubmed-article:3126396 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3126396 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3126396 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:3126396 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:3126396 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3126396 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:3126396 | pubmed:issn | 0028-0836 | lld:pubmed |
| pubmed-article:3126396 | pubmed:author | pubmed-author:WhiteJJ | lld:pubmed |
| pubmed-article:3126396 | pubmed:author | pubmed-author:KapplerJ WJW | lld:pubmed |
| pubmed-article:3126396 | pubmed:author | pubmed-author:MarrackP CPC | lld:pubmed |
| pubmed-article:3126396 | pubmed:author | pubmed-author:StaerzUU | lld:pubmed |
| pubmed-article:3126396 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3126396 | pubmed:day | 3 | lld:pubmed |
| pubmed-article:3126396 | pubmed:volume | 332 | lld:pubmed |
| pubmed-article:3126396 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3126396 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3126396 | pubmed:pagination | 35-40 | lld:pubmed |
| pubmed-article:3126396 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:3126396 | pubmed:meshHeading | pubmed-meshheading:3126396-... | lld:pubmed |
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| pubmed-article:3126396 | pubmed:meshHeading | pubmed-meshheading:3126396-... | lld:pubmed |
| pubmed-article:3126396 | pubmed:year | 1988 | lld:pubmed |
| pubmed-article:3126396 | pubmed:articleTitle | Self-tolerance eliminates T cells specific for Mls-modified products of the major histocompatibility complex. | lld:pubmed |
| pubmed-article:3126396 | pubmed:affiliation | Howard Hughes Medical Institute, Denver, Colorado. | lld:pubmed |
| pubmed-article:3126396 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3126396 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:3126396 | pubmed:publicationType | Review | lld:pubmed |
| pubmed-article:3126396 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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