| pubmed-article:3039291 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3039291 | lifeskim:mentions | umls-concept:C1257890 | lld:lifeskim |
| pubmed-article:3039291 | lifeskim:mentions | umls-concept:C1861263 | lld:lifeskim |
| pubmed-article:3039291 | lifeskim:mentions | umls-concept:C0599773 | lld:lifeskim |
| pubmed-article:3039291 | lifeskim:mentions | umls-concept:C1705914 | lld:lifeskim |
| pubmed-article:3039291 | lifeskim:mentions | umls-concept:C0079411 | lld:lifeskim |
| pubmed-article:3039291 | lifeskim:mentions | umls-concept:C0075290 | lld:lifeskim |
| pubmed-article:3039291 | pubmed:issue | 2-3 | lld:pubmed |
| pubmed-article:3039291 | pubmed:dateCreated | 1987-9-22 | lld:pubmed |
| pubmed-article:3039291 | pubmed:abstractText | M protein, a major surface protein and virulence factor for the group A streptococcus, exhibits extraordinary size variation in strains of the same serotype (Fischetti et al. 1985). RNA sequence analysis of spontaneous M protein size variants shows that deletion mutations arise in a single strain by homologous recombination events between intragenic tandem repeats. Similar deletion and duplication events also occur in serial streptococcal isolates from a single patient and among related strains in a recent outbreak. We discuss how homologous recombination events can lead to the generation of antigenic variation. | lld:pubmed |
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| pubmed-article:3039291 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3039291 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3039291 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:3039291 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3039291 | pubmed:month | May | lld:pubmed |
| pubmed-article:3039291 | pubmed:issn | 0026-8925 | lld:pubmed |
| pubmed-article:3039291 | pubmed:author | pubmed-author:ScottJ RJR | lld:pubmed |
| pubmed-article:3039291 | pubmed:author | pubmed-author:FischettiV... | lld:pubmed |
| pubmed-article:3039291 | pubmed:author | pubmed-author:HollingsheadS... | lld:pubmed |
| pubmed-article:3039291 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3039291 | pubmed:volume | 207 | lld:pubmed |
| pubmed-article:3039291 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3039291 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3039291 | pubmed:pagination | 196-203 | lld:pubmed |
| pubmed-article:3039291 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:3039291 | pubmed:year | 1987 | lld:pubmed |
| pubmed-article:3039291 | pubmed:articleTitle | Size variation in group A streptococcal M protein is generated by homologous recombination between intragenic repeats. | lld:pubmed |
| pubmed-article:3039291 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3039291 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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