| pubmed-article:3003163 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3003163 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:3003163 | lifeskim:mentions | umls-concept:C0224522 | lld:lifeskim |
| pubmed-article:3003163 | lifeskim:mentions | umls-concept:C0021755 | lld:lifeskim |
| pubmed-article:3003163 | lifeskim:mentions | umls-concept:C0012472 | lld:lifeskim |
| pubmed-article:3003163 | lifeskim:mentions | umls-concept:C0162745 | lld:lifeskim |
| pubmed-article:3003163 | lifeskim:mentions | umls-concept:C1527200 | lld:lifeskim |
| pubmed-article:3003163 | lifeskim:mentions | umls-concept:C0033268 | lld:lifeskim |
| pubmed-article:3003163 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:3003163 | pubmed:dateCreated | 1986-3-26 | lld:pubmed |
| pubmed-article:3003163 | pubmed:abstractText | The pathogenesis and progression of rheumatoid arthritis involves the production of biologically active lymphokines and monokines. Of these, interleukin 1 (IL-1) has been somewhat of a controversial molecule because it seems to evoke various biological responses in several different tissues. In these studies we demonstrate that three biological properties of human monocyte-derived IL-1 (T-lymphocyte activation and human synovial cell prostaglandin E2 and collagenase production) co-purify. The complementary DNA for the prominent pI 7 form of human IL-1 was expressed, purified, and tested. Any controversy now appears resolved since homogeneous recombinant human IL-1 stimulates prostaglandin E2 and collagenase from human synovial cells as well as activates T cells in vitro. | lld:pubmed |
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| pubmed-article:3003163 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:3003163 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:3003163 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3003163 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:3003163 | pubmed:issn | 0021-9738 | lld:pubmed |
| pubmed-article:3003163 | pubmed:author | pubmed-author:DinarelloC... | lld:pubmed |
| pubmed-article:3003163 | pubmed:author | pubmed-author:DayerJ MJM | lld:pubmed |
| pubmed-article:3003163 | pubmed:author | pubmed-author:DemczukSS | lld:pubmed |
| pubmed-article:3003163 | pubmed:author | pubmed-author:BurrusBB | lld:pubmed |
| pubmed-article:3003163 | pubmed:author | pubmed-author:de... | lld:pubmed |
| pubmed-article:3003163 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3003163 | pubmed:volume | 77 | lld:pubmed |
| pubmed-article:3003163 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3003163 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3003163 | pubmed:pagination | 645-8 | lld:pubmed |
| pubmed-article:3003163 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3003163 | pubmed:year | 1986 | lld:pubmed |
| pubmed-article:3003163 | pubmed:articleTitle | Human recombinant interleukin 1 stimulates collagenase and prostaglandin E2 production by human synovial cells. | lld:pubmed |
| pubmed-article:3003163 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3003163 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:3003163 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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