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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
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pubmed:dateCreated |
1986-2-13
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pubmed:abstractText |
Virus clones which express glycoprotein gC (gC+) were obtained from two persistently infected (p.i.) MDBK cell lines which had been independently established by infection with HSV-1(MP)10311, a gC- syncytial (syn) variant of herpes simplex virus type 1 strain MP [HSV-1(MP)]. The gC+ revertants were syn in MDBK, HEp-2, and Vero cell lines and in primary human fibroblasts; this offers further evidence that glycoprotein gC does not inhibit cell fusion. The gC+ revertants represented from 70 to 100 percent of the virions present in the virus populations examined, thus suggesting a possible selective advantage of the gC+ revertants in this system of persistent infection.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Mar
|
pubmed:issn |
0042-6822
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
141
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
306-10
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:3002019-Animals,
pubmed-meshheading:3002019-Cell Fusion,
pubmed-meshheading:3002019-Cell Line,
pubmed-meshheading:3002019-Cells, Cultured,
pubmed-meshheading:3002019-Cytopathogenic Effect, Viral,
pubmed-meshheading:3002019-Dogs,
pubmed-meshheading:3002019-Genes,
pubmed-meshheading:3002019-Genes, Viral,
pubmed-meshheading:3002019-Humans,
pubmed-meshheading:3002019-Kidney,
pubmed-meshheading:3002019-Mutation,
pubmed-meshheading:3002019-Phenotype,
pubmed-meshheading:3002019-Simplexvirus,
pubmed-meshheading:3002019-Viral Envelope Proteins,
pubmed-meshheading:3002019-Viral Plaque Assay,
pubmed-meshheading:3002019-Viral Proteins
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pubmed:year |
1985
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pubmed:articleTitle |
Characterization of virus obtained from MDBK cells persistently infected with a variant of herpes simplex virus type 1 strain MP [HSV-1(MP)].
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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