| pubmed-article:2972166 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C0205476 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C0020632 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C0023610 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C0243192 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C1446409 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C0243161 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C0015744 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C2911691 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C0205374 | lld:lifeskim |
| pubmed-article:2972166 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:2972166 | pubmed:dateCreated | 1988-11-8 | lld:pubmed |
| pubmed-article:2972166 | pubmed:abstractText | The hypothalamic pituitary axis was studied in patients with an abnormal pattern of gonadotropin release during chronic treatment with LH-RH agonist. Two patients had PCOD and the third demonstrated the early luteinization phenomenon. Following a well-defined gonadotropin rise with initiation of LH-RH treatment, no further response was noted. Stabilization of the LH:FSH ratio in PCOD patients was noted after 4 weeks of treatment. Administration of both native LH-RH (100 micrograms) and intravenous pulsatile LH-RH did not evoke any rise in LH. In addition to the above LH-RH challenges, the positive feedback was examined by administration of estradiol benzoate (EB). The study demonstrated that, although the pituitary did not respond to any LH-RH challenge, it may still respond by a rise in LH following EB administration. Both functions of the hypothalamic pituitary axis should be examined in order to determine the state of medical hypophysectomy. | lld:pubmed |
| pubmed-article:2972166 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2972166 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2972166 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2972166 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2972166 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2972166 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2972166 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2972166 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2972166 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2972166 | pubmed:author | pubmed-author:LunenfeldBB | lld:pubmed |
| pubmed-article:2972166 | pubmed:author | pubmed-author:BlanksteinJJ | lld:pubmed |
| pubmed-article:2972166 | pubmed:author | pubmed-author:BahariCC | lld:pubmed |
| pubmed-article:2972166 | pubmed:author | pubmed-author:ParienteCC | lld:pubmed |
| pubmed-article:2972166 | pubmed:author | pubmed-author:KokiaEE | lld:pubmed |
| pubmed-article:2972166 | pubmed:author | pubmed-author:ShadmiA LAL | lld:pubmed |
| pubmed-article:2972166 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2972166 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2972166 | pubmed:pagination | 1-11 | lld:pubmed |
| pubmed-article:2972166 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:meshHeading | pubmed-meshheading:2972166-... | lld:pubmed |
| pubmed-article:2972166 | pubmed:articleTitle | Abolishment of the positive feedback mechanism: a criterion for temporary medical hypophysectomy by LH-RH agonist. | lld:pubmed |
| pubmed-article:2972166 | pubmed:affiliation | Department of Obstetrics, Gynecology and Endocrinology, Chaim Sheba Medical Center, Sackler Medical School, Tel-Aviv University, Tel-Hashomer, Israel. | lld:pubmed |
| pubmed-article:2972166 | pubmed:publicationType | Journal Article | lld:pubmed |
