| pubmed-article:2968436 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2968436 | lifeskim:mentions | umls-concept:C0021289 | lld:lifeskim |
| pubmed-article:2968436 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:2968436 | lifeskim:mentions | umls-concept:C0330390 | lld:lifeskim |
| pubmed-article:2968436 | lifeskim:mentions | umls-concept:C0221102 | lld:lifeskim |
| pubmed-article:2968436 | lifeskim:mentions | umls-concept:C0003320 | lld:lifeskim |
| pubmed-article:2968436 | lifeskim:mentions | umls-concept:C1817959 | lld:lifeskim |
| pubmed-article:2968436 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:2968436 | pubmed:dateCreated | 1988-8-3 | lld:pubmed |
| pubmed-article:2968436 | pubmed:abstractText | The cellular basis of neonatally induced T cell tolerance has been investigated in a model system in which usage of a particular TCR V beta segment (V beta 6) is strongly correlated with reactivity to antigens encoded by the Mlsa genetic locus. Expression of V beta 6 by peripheral T cells was virtually abolished in BALB/c (H-2d, Mlsb) mice rendered neonatally tolerant to DBA/2 (H-2d, Mlsa) lymphoid cells, whereas control V beta 8-bearing T cells remained at near normal levels. Further analysis revealed that elimination of V beta 6+ T cells occurred in the thymus of neonatally tolerant mice and could not be explained by receptor modulation or T cell chimerism. These data thus support the clonal deletion model of tolerance induction. | lld:pubmed |
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| pubmed-article:2968436 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2968436 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2968436 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2968436 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:2968436 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:2968436 | pubmed:author | pubmed-author:MacDonaldH... | lld:pubmed |
| pubmed-article:2968436 | pubmed:author | pubmed-author:SchneiderRR | lld:pubmed |
| pubmed-article:2968436 | pubmed:author | pubmed-author:ZinkernagelR... | lld:pubmed |
| pubmed-article:2968436 | pubmed:author | pubmed-author:MONTIVV | lld:pubmed |
| pubmed-article:2968436 | pubmed:author | pubmed-author:HengartnerHH | lld:pubmed |
| pubmed-article:2968436 | pubmed:author | pubmed-author:PedrazziniTT | lld:pubmed |
| pubmed-article:2968436 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2968436 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:2968436 | pubmed:volume | 167 | lld:pubmed |
| pubmed-article:2968436 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2968436 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2968436 | pubmed:pagination | 2005-10 | lld:pubmed |
| pubmed-article:2968436 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2968436 | pubmed:year | 1988 | lld:pubmed |
| pubmed-article:2968436 | pubmed:articleTitle | Intrathymic elimination of Mlsa-reactive (V beta 6+) cells during neonatal tolerance induction to Mlsa-encoded antigens. | lld:pubmed |
| pubmed-article:2968436 | pubmed:affiliation | Ludwig Institute for Cancer Research, Lausanne Branch, Epalinges, Switzerland. | lld:pubmed |
| pubmed-article:2968436 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2968436 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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