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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
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pubmed:dateCreated |
1989-4-21
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pubmed:abstractText |
Enteropathogenic E. coli (EPEC) infection of Hep-2 cells proceeds through bacterial attachment to cell surface and internalization of adhered bacteria. EPEC attachment is a prerequisite for cell infection and is mediated by adhesins that recognize carbohydrate-containing receptors on cell membrane. Such endocytosis-inducer adhesins (EIA) also promote EPEC binding to infant enterocytes, suggesting that EIA may have an important role on EPEC gastroenteritis.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:issn |
0074-0276
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
82
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
87-90
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:2907090-Adhesins, Escherichia coli,
pubmed-meshheading:2907090-Bacterial Adhesion,
pubmed-meshheading:2907090-Bacterial Outer Membrane Proteins,
pubmed-meshheading:2907090-Diarrhea, Infantile,
pubmed-meshheading:2907090-Endocytosis,
pubmed-meshheading:2907090-Escherichia coli,
pubmed-meshheading:2907090-Humans,
pubmed-meshheading:2907090-Infant,
pubmed-meshheading:2907090-Virulence
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pubmed:articleTitle |
Endocytosis-inducer adhesins produced by enteropathogenic serogroups of Escherichia coli participate on bacterial attachment to infant enterocytes.
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pubmed:affiliation |
Serviço de Microbiologia e Imunologia, Faculdade de Ciências Médicas. UERJ. Rua Manuel de Abreu, Rio de Janeiro, Brasil.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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