Linked Life Data

2859472

Source:http://linkedlifedata.com/resource/pubmed/id/2859472

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
8436
pubmed:dateCreated
1985-6-6
pubmed:abstractText
The genetic contribution to schizophrenia is widely accepted, yet none of the proposed models of transmission has been convincing. Schizophrenia is generally viewed as aetiologically homogeneous with the exception of supposedly rare "phenocopies" associated with organic brain lesions and without a family history. However, up to one-third of schizophrenics have enlarged cerebral ventricles, and this appears to be a consequence of environmental damage. Although the aetiology of schizophrenia comprises genetic and environmental components acting in variable proportions, a simple division into familial and sporadic cases would facilitate research. Families with several ill members will be most valuable for molecular genetic studies, while the new brain imaging techniques should be particularly directed towards sporadic cases.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
0140-6736
pubmed:author
pubmed:issnType
Print
pubmed:day
4
pubmed:volume
1
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1023-6
pubmed:dateRevised
2004-11-17
pubmed:meshHeading
pubmed:year
1985
pubmed:articleTitle
Towards an aetiological classification of schizophrenia.
pubmed:publicationType
Journal Article, Review