| pubmed-article:2614572 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2614572 | lifeskim:mentions | umls-concept:C0079488 | lld:lifeskim |
| pubmed-article:2614572 | lifeskim:mentions | umls-concept:C0021368 | lld:lifeskim |
| pubmed-article:2614572 | lifeskim:mentions | umls-concept:C0439849 | lld:lifeskim |
| pubmed-article:2614572 | lifeskim:mentions | umls-concept:C1704242 | lld:lifeskim |
| pubmed-article:2614572 | lifeskim:mentions | umls-concept:C1261322 | lld:lifeskim |
| pubmed-article:2614572 | lifeskim:mentions | umls-concept:C0039225 | lld:lifeskim |
| pubmed-article:2614572 | lifeskim:mentions | umls-concept:C0293352 | lld:lifeskim |
| pubmed-article:2614572 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:2614572 | pubmed:dateCreated | 1990-3-13 | lld:pubmed |
| pubmed-article:2614572 | pubmed:abstractText | Gastric antral endoscopic pinch biopsies from a group of dyspeptic patients were analysed for acute and chronic inflammatory cell numbers in the lamina propria and surface epithelial layer using computer-linked graphic tablet planimetry, and independently graded for Campylobacter pylori (CP) infection using a visual scoring system with grade 1 assessed as patchy epithelial infection and grade 2 as a continuous layer of organisms on the mucosal surface extending into gastric pits. The study group consisted of 36 patients (18 duodenitis; 18 non-ulcer dyspepsia). Within the 140 biopsies analysed, grade 1 and 2 biopsies had significantly higher acute and chronic inflammatory cell counts than CP-negative biopsies (grade 0) in lamina and surface epithelium (P less than 0.001). Acute inflammatory cell counts were significantly higher in the surface epithelium in grade 1 (P less than 0.05) and grade 2 biopsies (P less than 0.001) but chronic inflammatory cells were only higher in grade 1 (P less than 0.01). No significant differences were present between grade 1 and grade 2 biopsies for any parameter. This study confirms that highly significant quantitative differences in the inflammatory status are related to the presence of CP. | lld:pubmed |
| pubmed-article:2614572 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2614572 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2614572 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2614572 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2614572 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:2614572 | pubmed:issn | 0022-3417 | lld:pubmed |
| pubmed-article:2614572 | pubmed:author | pubmed-author:SloanJ MJM | lld:pubmed |
| pubmed-article:2614572 | pubmed:author | pubmed-author:CollinsJ SJS | lld:pubmed |
| pubmed-article:2614572 | pubmed:author | pubmed-author:LoveA HAH | lld:pubmed |
| pubmed-article:2614572 | pubmed:author | pubmed-author:WebbM JMJ | lld:pubmed |
| pubmed-article:2614572 | pubmed:author | pubmed-author:HamiltonP WPW | lld:pubmed |
| pubmed-article:2614572 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2614572 | pubmed:volume | 159 | lld:pubmed |
| pubmed-article:2614572 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2614572 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2614572 | pubmed:pagination | 281-5 | lld:pubmed |
| pubmed-article:2614572 | pubmed:dateRevised | 2004-11-17 | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:meshHeading | pubmed-meshheading:2614572-... | lld:pubmed |
| pubmed-article:2614572 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2614572 | pubmed:articleTitle | Investigation of the relationship between gastric antral inflammation and Campylobacter pylori using graphic tablet planimetry. | lld:pubmed |
| pubmed-article:2614572 | pubmed:affiliation | Department of Medicine, Queen's University, Belfast, Northern Ireland. | lld:pubmed |
| pubmed-article:2614572 | pubmed:publicationType | Journal Article | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2614572 | lld:pubmed |
