| pubmed-article:2578888 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2578888 | lifeskim:mentions | umls-concept:C0006675 | lld:lifeskim |
| pubmed-article:2578888 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:2578888 | lifeskim:mentions | umls-concept:C1523873 | lld:lifeskim |
| pubmed-article:2578888 | lifeskim:mentions | umls-concept:C1948023 | lld:lifeskim |
| pubmed-article:2578888 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:2578888 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:2578888 | pubmed:dateCreated | 1985-4-24 | lld:pubmed |
| pubmed-article:2578888 | pubmed:abstractText | Three monoclonal antibodies selected for their recognition of parts of the T3-T cell receptor complex on human T lymphocytes were found to induce an increase in cytoplasmic free Ca2+ (Ca2+i) in the T cell leukemia line HPB-ALL as measured by Quin2 fluorescence. These reagents are directed against T3 (OKT3), a nonvariable T3-associated structure (WT-31) and the variable region of the T3-associated antigen receptor (T40/25) of this cell line. The rise in Ca2+i was dependent on the presence of extracellular Ca2+, occurred within 30 sec of stimulation, and was sustained for at least 10 min. Fab fragments of OKT3 also caused a rapid increase in Ca2+i, indicating that cross-linking is not necessary to induce a Ca2+ response. Alterations in plasma membrane potential and La3+ blocked the Ca2+ influx induced by OKT3 and T40/25. These data suggest that the T3-T cell receptor complex of human T lymphocytes may be an antigen-regulated Ca2+ channel. | lld:pubmed |
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| pubmed-article:2578888 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2578888 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2578888 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2578888 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:2578888 | pubmed:issn | 0092-8674 | lld:pubmed |
| pubmed-article:2578888 | pubmed:author | pubmed-author:TerhorstCC | lld:pubmed |
| pubmed-article:2578888 | pubmed:author | pubmed-author:CantleyL CLC | lld:pubmed |
| pubmed-article:2578888 | pubmed:author | pubmed-author:RosoffP MPM | lld:pubmed |
| pubmed-article:2578888 | pubmed:author | pubmed-author:OettgenH CHC | lld:pubmed |
| pubmed-article:2578888 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2578888 | pubmed:volume | 40 | lld:pubmed |
| pubmed-article:2578888 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2578888 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2578888 | pubmed:pagination | 583-90 | lld:pubmed |
| pubmed-article:2578888 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:2578888 | pubmed:year | 1985 | lld:pubmed |
| pubmed-article:2578888 | pubmed:articleTitle | Stimulation of the T3-T cell receptor complex induces a membrane-potential-sensitive calcium influx. | lld:pubmed |
| pubmed-article:2578888 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2578888 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:2578888 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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