| pubmed-article:2578432 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2578432 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:2578432 | lifeskim:mentions | umls-concept:C0038856 | lld:lifeskim |
| pubmed-article:2578432 | lifeskim:mentions | umls-concept:C0332157 | lld:lifeskim |
| pubmed-article:2578432 | lifeskim:mentions | umls-concept:C0041625 | lld:lifeskim |
| pubmed-article:2578432 | lifeskim:mentions | umls-concept:C0301625 | lld:lifeskim |
| pubmed-article:2578432 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:2578432 | pubmed:dateCreated | 1985-3-19 | lld:pubmed |
| pubmed-article:2578432 | pubmed:abstractText | Application of a contact-sensitizing agent to the skin of mice previously exposed to UV radiation at a different site results in the induction of hapten-specific suppressor T lymphocytes. When splenic lymphocytes from such mice were cultured with normal lymphocytes and hapten-conjugated splenic adherent cells, the primary proliferative response was suppressed. The cell responsible for the suppression in vitro was a T lymphocyte, and two signals were required for its induction, ultraviolet radiation and hapten sensitization. The T cell suppressing lymphoproliferation was specific for the hapten applied after UV radiation. The UV-induced T suppressor cell inhibited only primary lymphoproliferation; the response of lymphocytes from immunized mice was unaffected. The activity of the UV-induced suppressor cell was not affected by mitomycin C treatment. Thus, suppression of the primary proliferative response of lymphocytes to hapten-modified syngeneic cells in vitro correlates with in vivo suppression of contact hypersensitivity by these UV-induced suppressor cells. This suggests that the suppressor cells act by preventing the proliferation of hapten-specific responder clones. Use of this in vitro assay system should facilitate investigation of the characteristics of these cells and the mechanism by which these regulatory T lymphocytes inhibit contact sensitization. | lld:pubmed |
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| pubmed-article:2578432 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2578432 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2578432 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2578432 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:2578432 | pubmed:issn | 0019-2805 | lld:pubmed |
| pubmed-article:2578432 | pubmed:author | pubmed-author:UllrichS ESE | lld:pubmed |
| pubmed-article:2578432 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2578432 | pubmed:volume | 54 | lld:pubmed |
| pubmed-article:2578432 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2578432 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2578432 | pubmed:pagination | 343-52 | lld:pubmed |
| pubmed-article:2578432 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2578432 | pubmed:year | 1985 | lld:pubmed |
| pubmed-article:2578432 | pubmed:articleTitle | Suppression of lymphoproliferation by hapten-specific suppressor T lymphocytes from mice exposed to ultraviolet radiation. | lld:pubmed |
| pubmed-article:2578432 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2578432 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:2578432 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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