| pubmed-article:2573878 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2573878 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:2573878 | lifeskim:mentions | umls-concept:C0041217 | lld:lifeskim |
| pubmed-article:2573878 | lifeskim:mentions | umls-concept:C0332597 | lld:lifeskim |
| pubmed-article:2573878 | lifeskim:mentions | umls-concept:C0040649 | lld:lifeskim |
| pubmed-article:2573878 | pubmed:issue | 21 | lld:pubmed |
| pubmed-article:2573878 | pubmed:dateCreated | 1989-12-27 | lld:pubmed |
| pubmed-article:2573878 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2573878 | pubmed:abstractText | The genes encoding procyclin, the major glycoprotein expressed on the surface of procyclic forms of Trypanosoma brucei, comprise a multigene family. It has previously been demonstrated that procyclin genes in cloned trypanosome strains from Kenya and Uganda show restriction fragment polymorphisms. A detailed study of the Kenyan strain 227 has revealed that procyclin genes are arranged in tandem at 3 distinct loci (Pro A, B and C) and that the polymorphism is due to the duplication of 1.3 kb in the Pro A locus, which has generated an additional procyclin gene. Northern blot analysis has shown that at least 2 loci are transcribed and that a minimum of 3 procyclin genes are expressed within a cloned line. The transcription of procyclin genes is resistant to 1 mg ml-1 alpha-amanitin, whereas that of the 5' flanking gene in the Pro A locus is sensitive. This observation suggests that the two genes form part of separate transcription units with a promoter between them. | lld:pubmed |
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| pubmed-article:2573878 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2573878 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2573878 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2573878 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:2573878 | pubmed:issn | 0305-1048 | lld:pubmed |
| pubmed-article:2573878 | pubmed:author | pubmed-author:KönigEE | lld:pubmed |
| pubmed-article:2573878 | pubmed:author | pubmed-author:DeliusHH | lld:pubmed |
| pubmed-article:2573878 | pubmed:author | pubmed-author:WilliamsR ORO | lld:pubmed |
| pubmed-article:2573878 | pubmed:author | pubmed-author:CarringtonMM | lld:pubmed |
| pubmed-article:2573878 | pubmed:author | pubmed-author:RoditiII | lld:pubmed |
| pubmed-article:2573878 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2573878 | pubmed:day | 11 | lld:pubmed |
| pubmed-article:2573878 | pubmed:volume | 17 | lld:pubmed |
| pubmed-article:2573878 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2573878 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2573878 | pubmed:pagination | 8727-39 | lld:pubmed |
| pubmed-article:2573878 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2573878 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2573878 | pubmed:articleTitle | Duplication and transcription of procyclin genes in Trypanosoma brucei. | lld:pubmed |
| pubmed-article:2573878 | pubmed:affiliation | Kernforschungszentrum Karlsruhe, Institut für Genetik und Toxikologie, Karlsruhe, FRG. | lld:pubmed |
| pubmed-article:2573878 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2573878 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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