| pubmed-article:2572793 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2572793 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:2572793 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
| pubmed-article:2572793 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:2572793 | lifeskim:mentions | umls-concept:C0205100 | lld:lifeskim |
| pubmed-article:2572793 | pubmed:issue | 8670 | lld:pubmed |
| pubmed-article:2572793 | pubmed:dateCreated | 1989-12-5 | lld:pubmed |
| pubmed-article:2572793 | pubmed:abstractText | NG monomethyl-L-arginine (L-NMMA), a specific inhibitor of the synthesis of endothelium-derived nitric oxide (NO), was infused into the brachial arteries of healthy volunteers to study the role of NO in the control of forearm blood flow. L-NMMA caused a 50% fall in basal blood flow and attenuated the dilator response to infused acetylcholine but not that to glyceryl trinitrate. These results indicate that the dilator action of endothelium-derived NO contributes to the control of basal and stimulated regional blood flow in man. Impairment of production of NO might account for the abnormalities in vascular reactivity that characterise a wide variety of disease states. | lld:pubmed |
| pubmed-article:2572793 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2572793 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2572793 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2572793 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:2572793 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2572793 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:2572793 | pubmed:issn | 0140-6736 | lld:pubmed |
| pubmed-article:2572793 | pubmed:author | pubmed-author:MoncadaSS | lld:pubmed |
| pubmed-article:2572793 | pubmed:author | pubmed-author:CollierJJ | lld:pubmed |
| pubmed-article:2572793 | pubmed:author | pubmed-author:VallancePP | lld:pubmed |
| pubmed-article:2572793 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2572793 | pubmed:day | 28 | lld:pubmed |
| pubmed-article:2572793 | pubmed:volume | 2 | lld:pubmed |
| pubmed-article:2572793 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2572793 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2572793 | pubmed:pagination | 997-1000 | lld:pubmed |
| pubmed-article:2572793 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:2572793 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2572793 | pubmed:articleTitle | Effects of endothelium-derived nitric oxide on peripheral arteriolar tone in man. | lld:pubmed |
| pubmed-article:2572793 | pubmed:affiliation | Department of Pharmacology and Clinical Pharmacology, St George's Hospital Medical School, London. | lld:pubmed |
| pubmed-article:2572793 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2572793 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:2572793 | pubmed:publicationType | Controlled Clinical Trial | lld:pubmed |
| pubmed-article:2572793 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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