Statements in which the resource exists.
SubjectPredicateObjectContext
pubmed-article:2539769rdf:typepubmed:Citationlld:pubmed
pubmed-article:2539769lifeskim:mentionsumls-concept:C0086418lld:lifeskim
pubmed-article:2539769lifeskim:mentionsumls-concept:C0006826lld:lifeskim
pubmed-article:2539769lifeskim:mentionsumls-concept:C0314657lld:lifeskim
pubmed-article:2539769lifeskim:mentionsumls-concept:C1866227lld:lifeskim
pubmed-article:2539769lifeskim:mentionsumls-concept:C0220898lld:lifeskim
pubmed-article:2539769pubmed:issue1lld:pubmed
pubmed-article:2539769pubmed:dateCreated1989-5-23lld:pubmed
pubmed-article:2539769pubmed:abstractTextWe herein propose that specific chromosomal aberrations associated with specific neoplasms are not necessarily inherited as a constitutional trait. High-risk individuals may have a tendency to undergo specific somatic mutations or chromosomal rearrangements at a relatively low frequency in all tissues as mosaicism. This may potentiate the development of specific somatic cancers. Our hypothesis may be applicable to both hereditary and sporadic types of neoplasms.lld:pubmed
pubmed-article:2539769pubmed:granthttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:2539769pubmed:languageenglld:pubmed
pubmed-article:2539769pubmed:journalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:2539769pubmed:citationSubsetIMlld:pubmed
pubmed-article:2539769pubmed:statusMEDLINElld:pubmed
pubmed-article:2539769pubmed:issn0250-7005lld:pubmed
pubmed-article:2539769pubmed:authorpubmed-author:PathakSSlld:pubmed
pubmed-article:2539769pubmed:authorpubmed-author:HopwoodV LVLlld:pubmed
pubmed-article:2539769pubmed:authorpubmed-author:DhaliwalM KMKlld:pubmed
pubmed-article:2539769pubmed:issnTypePrintlld:pubmed
pubmed-article:2539769pubmed:volume9lld:pubmed
pubmed-article:2539769pubmed:ownerNLMlld:pubmed
pubmed-article:2539769pubmed:authorsCompleteYlld:pubmed
pubmed-article:2539769pubmed:pagination17-9lld:pubmed
pubmed-article:2539769pubmed:dateRevised2007-11-14lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:meshHeadingpubmed-meshheading:2539769-...lld:pubmed
pubmed-article:2539769pubmed:articleTitleGenetic susceptibility, somatic mosaicisms and predisposition to human cancer.lld:pubmed
pubmed-article:2539769pubmed:affiliationDepartment of Cell Biology, University of Texas M.D. Anderson Cancer Center, Houston 77030.lld:pubmed
pubmed-article:2539769pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:2539769pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
pubmed-article:2539769pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed