| pubmed-article:2532142 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2532142 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:2532142 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:2532142 | lifeskim:mentions | umls-concept:C0034790 | lld:lifeskim |
| pubmed-article:2532142 | lifeskim:mentions | umls-concept:C2347338 | lld:lifeskim |
| pubmed-article:2532142 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:2532142 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:2532142 | pubmed:dateCreated | 1990-1-30 | lld:pubmed |
| pubmed-article:2532142 | pubmed:abstractText | Herein we have investigated the ability of antigen to induce thymocyte death by apoptosis on the basis that this may be the mechanism for the deletion of autoreactive cells during T cell development. We show that the ability of the bacterial "superantigen" staphylococcal enterotoxin B to cause specific depletion of V beta 8+ cells when added to thymus organ cultures is accompanied by DNA degradation into oligonucleosomal fragments, indicating that depletion involves apoptosis. Our results provide the first direct evidence that antigen-induced apoptosis can be triggered in developing T cells. | lld:pubmed |
| pubmed-article:2532142 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2532142 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2532142 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2532142 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2532142 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:2532142 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2532142 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:2532142 | pubmed:issn | 0014-2980 | lld:pubmed |
| pubmed-article:2532142 | pubmed:author | pubmed-author:OwenJ JJJ | lld:pubmed |
| pubmed-article:2532142 | pubmed:author | pubmed-author:SmithC ACA | lld:pubmed |
| pubmed-article:2532142 | pubmed:author | pubmed-author:JenkinsonE... | lld:pubmed |
| pubmed-article:2532142 | pubmed:author | pubmed-author:WilliamsG TGT | lld:pubmed |
| pubmed-article:2532142 | pubmed:author | pubmed-author:KingstonRR | lld:pubmed |
| pubmed-article:2532142 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2532142 | pubmed:volume | 19 | lld:pubmed |
| pubmed-article:2532142 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2532142 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2532142 | pubmed:pagination | 2175-7 | lld:pubmed |
| pubmed-article:2532142 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:2532142 | pubmed:meshHeading | pubmed-meshheading:2532142-... | lld:pubmed |
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| pubmed-article:2532142 | pubmed:meshHeading | pubmed-meshheading:2532142-... | lld:pubmed |
| pubmed-article:2532142 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2532142 | pubmed:articleTitle | Antigen-induced apoptosis in developing T cells: a mechanism for negative selection of the T cell receptor repertoire. | lld:pubmed |
| pubmed-article:2532142 | pubmed:affiliation | Department of Anatomy, Medical School, University of Birmingham. | lld:pubmed |
| pubmed-article:2532142 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2532142 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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