| pubmed-article:2526388 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2526388 | lifeskim:mentions | umls-concept:C0019704 | lld:lifeskim |
| pubmed-article:2526388 | lifeskim:mentions | umls-concept:C0162596 | lld:lifeskim |
| pubmed-article:2526388 | lifeskim:mentions | umls-concept:C0242656 | lld:lifeskim |
| pubmed-article:2526388 | lifeskim:mentions | umls-concept:C0681842 | lld:lifeskim |
| pubmed-article:2526388 | lifeskim:mentions | umls-concept:C0205250 | lld:lifeskim |
| pubmed-article:2526388 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:2526388 | pubmed:dateCreated | 1989-8-14 | lld:pubmed |
| pubmed-article:2526388 | pubmed:abstractText | Levels of anticardiolipin antibodies (ACA) were measured in 55 patients with haemophilia A in serum samples obtained in 1983 and in 1987. Twenty-one patients were negative for anti HIV-1 antibodies in 1983 and remained negative in 1987; 34 patients had anti HIV-1 antibodies in 1983; 17 of these latter patients remained asymptomatic, whereas 17 patients developed ARC or AIDS during the 4 years follow-up. Thirteen anti HIV-1 negative patients had elevated ACA levels in 1983; subsequently, a significant decrease was observed in all these subjects (p less than 0.001). All anti HIV-1 positive patients had elevated ACA levels in 1983; normal values were found in 9 patients in 1987. Yet, these changes were not significant (p greater than 0.05). ACA levels were significantly higher in HIV-1 infected patients than in those without anti HIV-1 antibodies (p less than 0.05). There was no difference of ACA levels between the two anti HIV-1 positive patient groups, be it in 1983 or be it in 1987 (p greater than 0.05). There was no correlation of ACA levels with serum IgG concentrations, CD4+ lymphocytes, or the consumption of factor VIII concentrates. | lld:pubmed |
| pubmed-article:2526388 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2526388 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2526388 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2526388 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2526388 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2526388 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2526388 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2526388 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2526388 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:2526388 | pubmed:issn | 0340-6245 | lld:pubmed |
| pubmed-article:2526388 | pubmed:author | pubmed-author:HartlHH | lld:pubmed |
| pubmed-article:2526388 | pubmed:author | pubmed-author:LechnerKK | lld:pubmed |
| pubmed-article:2526388 | pubmed:author | pubmed-author:DudczakRR | lld:pubmed |
| pubmed-article:2526388 | pubmed:author | pubmed-author:PanzerSS | lld:pubmed |
| pubmed-article:2526388 | pubmed:author | pubmed-author:StainCC | lld:pubmed |
| pubmed-article:2526388 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2526388 | pubmed:day | 28 | lld:pubmed |
| pubmed-article:2526388 | pubmed:volume | 61 | lld:pubmed |
| pubmed-article:2526388 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2526388 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2526388 | pubmed:pagination | 81-5 | lld:pubmed |
| pubmed-article:2526388 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:2526388 | pubmed:meshHeading | pubmed-meshheading:2526388-... | lld:pubmed |
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| pubmed-article:2526388 | pubmed:meshHeading | pubmed-meshheading:2526388-... | lld:pubmed |
| pubmed-article:2526388 | pubmed:meshHeading | pubmed-meshheading:2526388-... | lld:pubmed |
| pubmed-article:2526388 | pubmed:meshHeading | pubmed-meshheading:2526388-... | lld:pubmed |
| pubmed-article:2526388 | pubmed:meshHeading | pubmed-meshheading:2526388-... | lld:pubmed |
| pubmed-article:2526388 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2526388 | pubmed:articleTitle | Anticardiolipin antibodies are elevated in HIV-1 infected haemophiliacs but do not predict for disease progression. | lld:pubmed |
| pubmed-article:2526388 | pubmed:affiliation | First Medical Clinic, University of Vienna, Austria. | lld:pubmed |
| pubmed-article:2526388 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2526388 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2526388 | lld:pubmed |
