pubmed-article:2499547 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:2499547 | lifeskim:mentions | umls-concept:C0032659 | lld:lifeskim |
pubmed-article:2499547 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:2499547 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
pubmed-article:2499547 | lifeskim:mentions | umls-concept:C1332714 | lld:lifeskim |
pubmed-article:2499547 | lifeskim:mentions | umls-concept:C0041221 | lld:lifeskim |
pubmed-article:2499547 | lifeskim:mentions | umls-concept:C1527148 | lld:lifeskim |
pubmed-article:2499547 | lifeskim:mentions | umls-concept:C0683598 | lld:lifeskim |
pubmed-article:2499547 | lifeskim:mentions | umls-concept:C0443348 | lld:lifeskim |
pubmed-article:2499547 | pubmed:issue | 7 | lld:pubmed |
pubmed-article:2499547 | pubmed:dateCreated | 1989-7-18 | lld:pubmed |
pubmed-article:2499547 | pubmed:abstractText | Mice injected with monoclonal antibody to the L3T4+ (CD4+) cell membrane surface glycoprotein of T lymphocytes and immunized with antigens of Trypanosoma cruzi had reduced antibody and cell-mediated immune responses to the organism. Mortality in these mice was 100% following challenge with 1.5 x 10(5) trypanosomes, whereas controls had a 50% survival rate, indicating that T-cell activation and viable L3T4+ (CD4+) T lymphocytes are essential for the development of resistance. | lld:pubmed |
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pubmed-article:2499547 | pubmed:language | eng | lld:pubmed |
pubmed-article:2499547 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2499547 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:2499547 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:2499547 | pubmed:month | Jul | lld:pubmed |
pubmed-article:2499547 | pubmed:issn | 0019-9567 | lld:pubmed |
pubmed-article:2499547 | pubmed:author | pubmed-author:AraujoF GFG | lld:pubmed |
pubmed-article:2499547 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:2499547 | pubmed:volume | 57 | lld:pubmed |
pubmed-article:2499547 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:2499547 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:2499547 | pubmed:pagination | 2246-8 | lld:pubmed |
pubmed-article:2499547 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:2499547 | pubmed:year | 1989 | lld:pubmed |
pubmed-article:2499547 | pubmed:articleTitle | Development of resistance to Trypanosoma cruzi in mice depends on a viable population of L3T4+ (CD4+) T lymphocytes. | lld:pubmed |
pubmed-article:2499547 | pubmed:affiliation | Department of Immunology and Infectious Diseases, Palo Alto Medical Foundation, California 94301. | lld:pubmed |
pubmed-article:2499547 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:2499547 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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