pubmed-article:2473304 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:2473304 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
pubmed-article:2473304 | lifeskim:mentions | umls-concept:C0079281 | lld:lifeskim |
pubmed-article:2473304 | lifeskim:mentions | umls-concept:C0003483 | lld:lifeskim |
pubmed-article:2473304 | lifeskim:mentions | umls-concept:C0441472 | lld:lifeskim |
pubmed-article:2473304 | lifeskim:mentions | umls-concept:C1521991 | lld:lifeskim |
pubmed-article:2473304 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
pubmed-article:2473304 | pubmed:dateCreated | 1989-8-21 | lld:pubmed |
pubmed-article:2473304 | pubmed:abstractText | The vasoconstricting action of endothelin-1 (ET-1) is partly mediated by voltage-dependent L-type Ca2+ channels. Activation of the Ca2+ channels is indirect. ET-1 action involves (i) the hydrolysis of phosphatidylinositol and the release of Ca2+ from internal stores and (ii) the opening of a nonselective cation channel in the plasma membrane. The resulting depolarization triggers the activity of L-type Ca2+ channels. | lld:pubmed |
pubmed-article:2473304 | pubmed:language | eng | lld:pubmed |
pubmed-article:2473304 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2473304 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:2473304 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2473304 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2473304 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2473304 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2473304 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:2473304 | pubmed:issn | 0160-2446 | lld:pubmed |
pubmed-article:2473304 | pubmed:author | pubmed-author:LazdunskiMM | lld:pubmed |
pubmed-article:2473304 | pubmed:author | pubmed-author:FrelinCC | lld:pubmed |
pubmed-article:2473304 | pubmed:author | pubmed-author:BarhaninJJ | lld:pubmed |
pubmed-article:2473304 | pubmed:author | pubmed-author:VignePP | lld:pubmed |
pubmed-article:2473304 | pubmed:author | pubmed-author:Schmid-Allian... | lld:pubmed |
pubmed-article:2473304 | pubmed:author | pubmed-author:Van... | lld:pubmed |
pubmed-article:2473304 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:2473304 | pubmed:volume | 13 Suppl 5 | lld:pubmed |
pubmed-article:2473304 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:2473304 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:2473304 | pubmed:pagination | S186-7 | lld:pubmed |
pubmed-article:2473304 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
pubmed-article:2473304 | pubmed:meshHeading | pubmed-meshheading:2473304-... | lld:pubmed |
pubmed-article:2473304 | pubmed:meshHeading | pubmed-meshheading:2473304-... | lld:pubmed |
pubmed-article:2473304 | pubmed:meshHeading | pubmed-meshheading:2473304-... | lld:pubmed |
pubmed-article:2473304 | pubmed:meshHeading | pubmed-meshheading:2473304-... | lld:pubmed |
pubmed-article:2473304 | pubmed:meshHeading | pubmed-meshheading:2473304-... | lld:pubmed |
pubmed-article:2473304 | pubmed:meshHeading | pubmed-meshheading:2473304-... | lld:pubmed |
pubmed-article:2473304 | pubmed:meshHeading | pubmed-meshheading:2473304-... | lld:pubmed |
pubmed-article:2473304 | pubmed:meshHeading | pubmed-meshheading:2473304-... | lld:pubmed |
pubmed-article:2473304 | pubmed:year | 1989 | lld:pubmed |
pubmed-article:2473304 | pubmed:articleTitle | Molecular mechanism of endothelin-1 action on aortic cells. | lld:pubmed |
pubmed-article:2473304 | pubmed:affiliation | Centre de Biochimie du Centre National de la Recherche Scientifique, Nice, France. | lld:pubmed |
pubmed-article:2473304 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:2473304 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2473304 | lld:pubmed |