| pubmed-article:2189006 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2189006 | lifeskim:mentions | umls-concept:C1265129 | lld:lifeskim |
| pubmed-article:2189006 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:2189006 | lifeskim:mentions | umls-concept:C1522618 | lld:lifeskim |
| pubmed-article:2189006 | lifeskim:mentions | umls-concept:C0041042 | lld:lifeskim |
| pubmed-article:2189006 | lifeskim:mentions | umls-concept:C0936012 | lld:lifeskim |
| pubmed-article:2189006 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:2189006 | pubmed:dateCreated | 1990-7-2 | lld:pubmed |
| pubmed-article:2189006 | pubmed:abstractText | Trimethoprim (TMP) resistance among Shigella species isolated from Finnish travelers increased from 3.0% in 1975-1982 to 42.0%-43.8% in 1987-1988. Of the 317 TMP-resistant Shigella isolates identified during 1975-1988, 175 (55%) collected in 1985-1987 and in 1988 were tested further. Almost all (98%) were highly resistant to TMP, suggesting a plasmid-mediated origin. The type I dihydrofolate reductase (DHFR) gene was detected in 85% of the isolates studied. Twenty-three percent of the type I DHFR-positive isolates failed to hybridize with a probe detecting only Tn7-derived sequences, suggesting that the type I DHFR gene may occur independently of transposon Tn7. Four of the five Shigella species isolated from travelers to Sri Lanka hybridized with the probe for type V DHFR gene, implying a local distribution of the type V DHFR gene. The type II and type III DHFR genes were not found among the isolates studied. Only 12% of the TMP-resistant Shigella isolates failed to hybridize with any of the DHFR gene probes used. | lld:pubmed |
| pubmed-article:2189006 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2189006 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2189006 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:2189006 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2189006 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2189006 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2189006 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:2189006 | pubmed:issn | 0022-1899 | lld:pubmed |
| pubmed-article:2189006 | pubmed:author | pubmed-author:SundströmLL | lld:pubmed |
| pubmed-article:2189006 | pubmed:author | pubmed-author:JahkolaMM | lld:pubmed |
| pubmed-article:2189006 | pubmed:author | pubmed-author:HeikkiläEE | lld:pubmed |
| pubmed-article:2189006 | pubmed:author | pubmed-author:HuovinenPP | lld:pubmed |
| pubmed-article:2189006 | pubmed:author | pubmed-author:SiitonenAA | lld:pubmed |
| pubmed-article:2189006 | pubmed:author | pubmed-author:FlingMM | lld:pubmed |
| pubmed-article:2189006 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2189006 | pubmed:volume | 161 | lld:pubmed |
| pubmed-article:2189006 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2189006 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2189006 | pubmed:pagination | 1242-8 | lld:pubmed |
| pubmed-article:2189006 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:2189006 | pubmed:meshHeading | pubmed-meshheading:2189006-... | lld:pubmed |
| pubmed-article:2189006 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2189006 | pubmed:articleTitle | Increase of trimethoprim resistance among Shigella species, 1975-1988: analysis of resistance mechanisms. | lld:pubmed |
| pubmed-article:2189006 | pubmed:affiliation | Department of Medical Microbiology, University of Turku, Finland. | lld:pubmed |
| pubmed-article:2189006 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2189006 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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