| pubmed-article:2184369 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C0019704 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C0020971 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C1332717 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C1706438 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C0039195 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C0085358 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C0017278 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C0085408 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C1422036 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C1413244 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C2698600 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:2184369 | lifeskim:mentions | umls-concept:C1998793 | lld:lifeskim |
| pubmed-article:2184369 | pubmed:issue | 6269 | lld:pubmed |
| pubmed-article:2184369 | pubmed:dateCreated | 1990-5-30 | lld:pubmed |
| pubmed-article:2184369 | pubmed:abstractText | To reduce the risks of immunization with killed or live attenuated virus vaccines, it may be advantageous to use a pure, defined antigen that contains determinants for both humoral and cellular immunity. However, although most non-living intact protein preparations induce antibodies and CD4+ major histocompatibility complex (MHC) class II-restricted helper and/or cytotoxic T lymphocytes (CTL), they do not elicit CD8+ MHC class I restricted CTL. Indeed, with a few exceptions, it has not so far been possible to induce CD8+ CTL by immunizing with intact soluble proteins. We show here that a single subcutaneous immunization in mice with immunostimulating complexes containing either purified intact gp160 envelope glycoprotein of the human immunodeficiency virus (HIV)-1 or influenza haemagglutinin results in reproducible and long-lasting priming of HIV specific or influenza-specific CD8+, MHC class I restricted CTL. | lld:pubmed |
| pubmed-article:2184369 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2184369 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2184369 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2184369 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2184369 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2184369 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:2184369 | pubmed:issn | 0028-0836 | lld:pubmed |
| pubmed-article:2184369 | pubmed:author | pubmed-author:TakahashiHH | lld:pubmed |
| pubmed-article:2184369 | pubmed:author | pubmed-author:MoreinBB | lld:pubmed |
| pubmed-article:2184369 | pubmed:author | pubmed-author:BerzofskyJ... | lld:pubmed |
| pubmed-article:2184369 | pubmed:author | pubmed-author:GermainR NRN | lld:pubmed |
| pubmed-article:2184369 | pubmed:author | pubmed-author:TakeshitaTT | lld:pubmed |
| pubmed-article:2184369 | pubmed:author | pubmed-author:PutneySS | lld:pubmed |
| pubmed-article:2184369 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2184369 | pubmed:day | 26 | lld:pubmed |
| pubmed-article:2184369 | pubmed:volume | 344 | lld:pubmed |
| pubmed-article:2184369 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2184369 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2184369 | pubmed:pagination | 873-5 | lld:pubmed |
| pubmed-article:2184369 | pubmed:dateRevised | 2003-11-14 | lld:pubmed |
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| pubmed-article:2184369 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2184369 | pubmed:articleTitle | Induction of CD8+ cytotoxic T cells by immunization with purified HIV-1 envelope protein in ISCOMs. | lld:pubmed |
| pubmed-article:2184369 | pubmed:affiliation | Molecular Immunogenetics and Vaccine Research Section, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892. | lld:pubmed |
| pubmed-article:2184369 | pubmed:publicationType | Journal Article | lld:pubmed |
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