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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2011-7-27
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pubmed:abstractText |
Excessive inflammatory host response increases morbidity and mortality associated with seasonal respiratory influenza, and highly pathogenic virus strains are characterized by massive infiltration of monocytes and/or macrophages that produce a storm of injurious cytokines.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
1537-6613
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:day |
15
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pubmed:volume |
204
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
592-600
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pubmed:dateRevised |
2011-10-11
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pubmed:meshHeading |
pubmed-meshheading:21791661-Animals,
pubmed-meshheading:21791661-Cell Death,
pubmed-meshheading:21791661-Cytokines,
pubmed-meshheading:21791661-Gene Expression Regulation,
pubmed-meshheading:21791661-Inflammation,
pubmed-meshheading:21791661-Influenza A Virus, H3N2 Subtype,
pubmed-meshheading:21791661-Lung,
pubmed-meshheading:21791661-Lung Diseases,
pubmed-meshheading:21791661-Mice,
pubmed-meshheading:21791661-Mice, Knockout,
pubmed-meshheading:21791661-Orthomyxoviridae Infections,
pubmed-meshheading:21791661-Serpins,
pubmed-meshheading:21791661-Time Factors
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pubmed:year |
2011
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pubmed:articleTitle |
Critical role of serpinB1 in regulating inflammatory responses in pulmonary influenza infection.
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pubmed:affiliation |
Immune Disease Institute and Program in Cellular and Molecular Medicine at Children's Hospital and Department of Pediatrics, Harvard Medical School, Boston, Massachusetts, USA.
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pubmed:publicationType |
Journal Article,
Research Support, N.I.H., Extramural
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