21788424

Source:http://linkedlifedata.com/resource/pubmed/id/21788424

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0014406, umls-concept:C0021270, umls-concept:C0021289, umls-concept:C0086582, umls-concept:C0220806, umls-concept:C0268301, umls-concept:C0332157, umls-concept:C0332240, umls-concept:C0678804, umls-concept:C1568245
pubmed:issue	4
pubmed:dateCreated	2011-9-29
pubmed:abstractText	46,XY disorders of sex differentiation (46,XY DSD) can be due to a testis determination defect, an androgen biosynthesis defect, or androgen resistance (complete or partial androgen insensitivity syndrome (PAIS), or 5? reductase deficiency). We aimed to evaluate the impact of a prenatal contamination by environmental xenoestrogens in 'idiopathic' PAIS-like phenotype.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9423848
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/DNA, http://linkedlifedata.com/resource/pubmed/chemical/Endocrine Disruptors, http://linkedlifedata.com/resource/pubmed/chemical/Environmental Pollutants, http://linkedlifedata.com/resource/pubmed/chemical/Estrogens, Non-Steroidal, http://linkedlifedata.com/resource/pubmed/chemical/Testosterone
pubmed:status	MEDLINE
pubmed:month	Oct
pubmed:issn	1479-683X
pubmed:author	pubmed-author:AudranFrançoiseF, pubmed-author:GaspariLauraL, pubmed-author:KalfaNicolasN, pubmed-author:MaïmounLaurentL, pubmed-author:OrsiniMatteaM, pubmed-author:ParisFrançoiseF, pubmed-author:PhilibertPascalP, pubmed-author:ServantNadègeN, pubmed-author:SultanCharlesC
pubmed:issnType	Electronic
pubmed:volume	165
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	579-87
pubmed:meshHeading	pubmed-meshheading:21788424-46, XY Disorders of Sex Development, pubmed-meshheading:21788424-Androgen-Insensitivity Syndrome, pubmed-meshheading:21788424-Biological Assay, pubmed-meshheading:21788424-Cryptorchidism, pubmed-meshheading:21788424-DNA, pubmed-meshheading:21788424-Endocrine Disruptors, pubmed-meshheading:21788424-Environmental Exposure, pubmed-meshheading:21788424-Environmental Pollutants, pubmed-meshheading:21788424-Estrogens, Non-Steroidal, pubmed-meshheading:21788424-Europe, pubmed-meshheading:21788424-Female, pubmed-meshheading:21788424-HeLa Cells, pubmed-meshheading:21788424-Humans, pubmed-meshheading:21788424-Hypospadias, pubmed-meshheading:21788424-Infant, pubmed-meshheading:21788424-Infant, Newborn, pubmed-meshheading:21788424-Male, pubmed-meshheading:21788424-Occupational Exposure, pubmed-meshheading:21788424-Pregnancy, pubmed-meshheading:21788424-Prenatal Exposure Delayed Effects, pubmed-meshheading:21788424-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:21788424-Risk Factors, pubmed-meshheading:21788424-Testosterone
pubmed:year	2011
pubmed:articleTitle	'Idiopathic' partial androgen insensitivity syndrome in 28 newborn and infant males: impact of prenatal exposure to environmental endocrine disruptor chemicals?
pubmed:affiliation	Unité d'Endocrinologie-Gynécologie Pédiatrique, Service de Pédiatrie 1, Hôpital Arnaud-de-Villeneuve, Montpellier, France.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't