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rdf:type |
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lifeskim:mentions |
umls-concept:C0026336,
umls-concept:C0035203,
umls-concept:C0036982,
umls-concept:C0199470,
umls-concept:C0205263,
umls-concept:C0205359,
umls-concept:C0242184,
umls-concept:C0333275,
umls-concept:C0871261,
umls-concept:C1155002,
umls-concept:C1704632,
umls-concept:C1706817,
umls-concept:C2911692
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pubmed:issue |
3
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pubmed:dateCreated |
2011-8-25
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pubmed:abstractText |
The interaction between neutrophils and activated endothelium is essential for the development of multiple organ dysfunction in patients with hemorrhagic shock (HS). Mechanical ventilation frequently is used in patients with HS. The authors sought to investigate the consequences of mechanical ventilation of mice subjected to HS on microvascular endothelial activation in the lung and kidney.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
1528-1175
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pubmed:author |
pubmed-author:AartsLeon P H JLP,
pubmed-author:HeeringaPeterP,
pubmed-author:HouwertjesMartin CMC,
pubmed-author:JongmanRianne MRM,
pubmed-author:MolemaGrietjeG,
pubmed-author:SchefferGert JanGJ,
pubmed-author:SchipperMartinM,
pubmed-author:TeppemaLuc JLJ,
pubmed-author:VanekerMichielM,
pubmed-author:WulfertFrancis MFM,
pubmed-author:ZijlstraJan GJG,
pubmed-author:van MeursMatijsM
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pubmed:issnType |
Electronic
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pubmed:volume |
115
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
474-82
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pubmed:meshHeading |
pubmed-meshheading:21778876-Animals,
pubmed-meshheading:21778876-Anoxia,
pubmed-meshheading:21778876-Antigens, CD31,
pubmed-meshheading:21778876-Chemokine CX3CL1,
pubmed-meshheading:21778876-E-Selectin,
pubmed-meshheading:21778876-Endothelial Cells,
pubmed-meshheading:21778876-Enzyme-Linked Immunosorbent Assay,
pubmed-meshheading:21778876-Gene Expression,
pubmed-meshheading:21778876-Immunohistochemistry,
pubmed-meshheading:21778876-Inflammation,
pubmed-meshheading:21778876-Intercellular Adhesion Molecule-1,
pubmed-meshheading:21778876-Male,
pubmed-meshheading:21778876-Mice,
pubmed-meshheading:21778876-Mice, Inbred C57BL,
pubmed-meshheading:21778876-Microcirculation,
pubmed-meshheading:21778876-Oxygen,
pubmed-meshheading:21778876-Pulmonary Circulation,
pubmed-meshheading:21778876-Renal Circulation,
pubmed-meshheading:21778876-Respiration, Artificial,
pubmed-meshheading:21778876-Respiratory Mechanics,
pubmed-meshheading:21778876-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:21778876-Shock, Hemorrhagic,
pubmed-meshheading:21778876-Vascular Cell Adhesion Molecule-1
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pubmed:year |
2011
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pubmed:articleTitle |
Hemorrhagic shock-induced endothelial cell activation in a spontaneous breathing and a mechanical ventilation hemorrhagic shock model is induced by a proinflammatory response and not by hypoxia.
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pubmed:affiliation |
Department of Pathology and Medical Biology, Medical Biology Section, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands. m.van.meurs@icv.umcg.nl
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pubmed:publicationType |
Journal Article
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