Source:http://linkedlifedata.com/resource/pubmed/id/21737639
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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
6
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| pubmed:dateCreated |
2011-7-8
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| pubmed:abstractText |
Chewing of regurgitated food elicits in baboons life-long gastro-esophageal reflux (GER). The acid reflux transforms the multilayered squamous cell epithelium of the esophagus into columnar-lined mucosa with mucus-producing accessory glands. The function of this mucous gland metaplasia (MGM), which mimics Barrett's mucosa with MGM in humans, is to buffer the gastric acid entering the esophagus during regurgitation. In a previous study of entire esophagi, the majority of baboons showed MGM. The gastric mucosa was not investigated.
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| pubmed:language |
eng
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| pubmed:journal | |
| pubmed:citationSubset |
IM
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| pubmed:status |
MEDLINE
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| pubmed:month |
Jun
|
| pubmed:issn |
1791-7530
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| pubmed:author | |
| pubmed:issnType |
Electronic
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| pubmed:volume |
31
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| pubmed:owner |
NLM
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| pubmed:authorsComplete |
Y
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| pubmed:pagination |
2187-90
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| pubmed:meshHeading | |
| pubmed:year |
2011
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| pubmed:articleTitle |
Mucous gland metaplasia in the esophagus and gastric mucosa in baboons.
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| pubmed:affiliation |
Southwest National Primate Research Center at the Southwest Foundation for Biomedical Research, San Antonio, TX, USA. Carlos.Rubio@ki.se
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| pubmed:publicationType |
Journal Article
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