21663621

Source:http://linkedlifedata.com/resource/pubmed/id/21663621

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0044602, umls-concept:C0205224, umls-concept:C1510411, umls-concept:C1533691, umls-concept:C1710082
pubmed:dateCreated	2011-7-7
pubmed:abstractText	High-risk human papillomavirus (hrHPV) infections are causally related to cervical cancer development. The additional (epi)genetic alterations driving malignant transformation of hrHPV-infected cells however, are not yet fully elucidated. In this study we experimentally assessed the role of the PI3-kinase pathway and its regulator PIK3CA, which is frequently altered in cervical cancer, in HPV-induced transformation.
pubmed:grant	http://linkedlifedata.com/resource/pubmed/grant/CA83679
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pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/101147698
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/2-(4-morpholinyl)-8-phenyl-4H-1-benz..., http://linkedlifedata.com/resource/pubmed/chemical/Chromones, http://linkedlifedata.com/resource/pubmed/chemical/Enzyme Inhibitors, http://linkedlifedata.com/resource/pubmed/chemical/Morpholines, http://linkedlifedata.com/resource/pubmed/chemical/PIK3CA protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Phosphatidylinositol 3-Kinases, http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger, http://linkedlifedata.com/resource/pubmed/chemical/RNA, Small Interfering
pubmed:status	MEDLINE
pubmed:issn	1476-4598
pubmed:author	pubmed-author:BanerjeeN SanjibNS, pubmed-author:BrokerThomas RTR, pubmed-author:ChowLouise TLT, pubmed-author:De-Castro ArceJohannaJ, pubmed-author:HenkenFlorianne EFE, pubmed-author:MeijerChris J L MCJ, pubmed-author:RöslFrankF, pubmed-author:SnijdersPeter J FPJ, pubmed-author:SteenbergenRenske D MRD
pubmed:issnType	Electronic
pubmed:volume	10
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	71
pubmed:meshHeading	pubmed-meshheading:21663621-Alphapapillomavirus, pubmed-meshheading:21663621-Carcinoma, pubmed-meshheading:21663621-Cell Differentiation, pubmed-meshheading:21663621-Cell Line, Transformed, pubmed-meshheading:21663621-Cell Movement, pubmed-meshheading:21663621-Cell Proliferation, pubmed-meshheading:21663621-Cell Survival, pubmed-meshheading:21663621-Cell Transformation, Neoplastic, pubmed-meshheading:21663621-Chromones, pubmed-meshheading:21663621-Enzyme Inhibitors, pubmed-meshheading:21663621-Female, pubmed-meshheading:21663621-Gene Expression Regulation, Neoplastic, pubmed-meshheading:21663621-Gene Silencing, pubmed-meshheading:21663621-Humans, pubmed-meshheading:21663621-Morpholines, pubmed-meshheading:21663621-Phosphatidylinositol 3-Kinases, pubmed-meshheading:21663621-RNA, Messenger, pubmed-meshheading:21663621-RNA, Small Interfering, pubmed-meshheading:21663621-Signal Transduction, pubmed-meshheading:21663621-Uterine Cervical Neoplasms
pubmed:year	2011
pubmed:articleTitle	PIK3CA-mediated PI3-kinase signalling is essential for HPV-induced transformation in vitro.
pubmed:affiliation	Department of Pathology, Unit of Molecular Pathology, VU University Medical Center, PO Box 7057, 1007 MB Amsterdam, The Netherlands.

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