Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
6
pubmed:dateCreated
2011-6-29
pubmed:abstractText
Reverse cholesterol transport (RCT) is a critical mechanism for the anti-atherogenic property of HDL. The inhibitory effect of the sulfonylurea agent (SUA) glibenclamide on ATP binding-cassette transporter (ABC) A1 may decrease HDL function but it remains unclear whether it attenuates RCT in vivo. We therefore investigated how the SUAs glibenclamide and glimepiride affected the functionality of ABCA1/ABCG1 and scavenger receptor class B type I (SR-BI) expression in macrophages in vitro and overall RCT in vivo.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
1880-3873
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
18
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
513-30
pubmed:meshHeading
pubmed-meshheading:21636950-ATP-Binding Cassette Transporters, pubmed-meshheading:21636950-Animals, pubmed-meshheading:21636950-Biological Transport, pubmed-meshheading:21636950-Blotting, Western, pubmed-meshheading:21636950-Cells, Cultured, pubmed-meshheading:21636950-Cholesterol, pubmed-meshheading:21636950-Cricetinae, pubmed-meshheading:21636950-Glyburide, pubmed-meshheading:21636950-HEK293 Cells, pubmed-meshheading:21636950-Humans, pubmed-meshheading:21636950-Hypoglycemic Agents, pubmed-meshheading:21636950-Liver, pubmed-meshheading:21636950-Macrophages, pubmed-meshheading:21636950-Macrophages, Peritoneal, pubmed-meshheading:21636950-Male, pubmed-meshheading:21636950-Mice, pubmed-meshheading:21636950-Mice, Inbred C57BL, pubmed-meshheading:21636950-RNA, Messenger, pubmed-meshheading:21636950-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:21636950-Scavenger Receptors, Class B
pubmed:year
2011
pubmed:articleTitle
Effect of sulfonylurea agents on reverse cholesterol transport in vitro and vivo.
pubmed:affiliation
Division of Anti-aging, Department of Internal Medicine, National Defense Medical College, Saitama, Japan.
pubmed:publicationType
Journal Article, In Vitro