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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
7
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pubmed:dateCreated |
2011-6-28
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pubmed:abstractText |
Cholecystokinin (CCK) is released in response to lipid intake and stimulates insulin secretion. We hypothesized that CCK deficiency would alter the regulation of insulin secretion and glucose homeostasis.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
1939-327X
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pubmed:author |
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pubmed:copyrightInfo |
© 2011 by the American Diabetes Association.
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pubmed:issnType |
Electronic
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pubmed:volume |
60
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
2000-7
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:21602512-Animals,
pubmed-meshheading:21602512-Arginine,
pubmed-meshheading:21602512-Cholecystokinin,
pubmed-meshheading:21602512-Diet, Fat-Restricted,
pubmed-meshheading:21602512-Dietary Fats,
pubmed-meshheading:21602512-Glucose,
pubmed-meshheading:21602512-Glucose Intolerance,
pubmed-meshheading:21602512-Insulin,
pubmed-meshheading:21602512-Insulin Resistance,
pubmed-meshheading:21602512-Insulin-Secreting Cells,
pubmed-meshheading:21602512-Mice,
pubmed-meshheading:21602512-Mice, Knockout
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pubmed:year |
2011
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pubmed:articleTitle |
Impaired insulin secretion and enhanced insulin sensitivity in cholecystokinin-deficient mice.
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pubmed:affiliation |
Cincinnati Obesity Research Center, Metabolic Diseases Institute, University of Cincinnati, Cincinnati, Ohio, USA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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