Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:dateCreated
2011-6-13
pubmed:abstractText
Environmental stress plays an important role in the development of glucose intolerance influencing lipid and glucose metabolism through sympathetic nervous system, cytokines and hormones such as glucocorticoids, catecholamines and glucagon. Otherwise, fish oil prevents glucose intolerance and insulin resistance. Although the mechanisms involved are not fully understood, it is known that sympathetic and HPA responses are blunted and catecholamines and glucocorticoids concentrations can be modulated by fish consumption. The aim of the present study was to evaluate whether fish oil, on a normal lipidic diet: 1) could prevent the effect of footshock-stress on the development of glucose intolerance; 2) modified adiponectin receptor and serum concentration; and 3) also modified TNF-?, IL-6 and interleukin-10 (IL-10) levels in adipose tissue and liver. The study was performed in thirty day-old male Wistar randomly assigned into four groups: no stressed (C) and stressed (CS) rats fed with control diet, and no stressed (F) and stressed (FS) rats fed with a fish oil rich diet. The stress was performed as a three daily footshock stress sessions.
pubmed:commentsCorrections
http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-10855527, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-10999830, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-11237197, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-11514845, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-11684087, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-12269788, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-12573522, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-12782233, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-12837849, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-14652354, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-14686961, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-15012619, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-15752770, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-15897474, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-15925295, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-16469141, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-16567512, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-16823476, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-16965635, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-17055120, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-17433709, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-17574233, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-18235842, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-18986529, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-19148125, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-19394055, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-19584720, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-19906806, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-20149625, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-20471407, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-3099779, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-6018, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-7691892, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-8477665, http://linkedlifedata.com/resource/pubmed/commentcorrection/21569357-9398739
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
1476-511X
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
10
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
71
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed:year
2011
pubmed:articleTitle
Fish oil consumption prevents glucose intolerance and hypercorticosteronemy in footshock-stressed rats.
pubmed:affiliation
Departamento de Fisiologia, Universidade de São Paulo - UNIFESP - Rua Botucatu, 862, 2nd floor, Edifício de Ciências Biomédicas, Vila Clementino, São Paulo, SP, Brazil.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't