rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
2
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pubmed:dateCreated |
2011-7-27
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pubmed:abstractText |
Recent research has shown netrin-1 to promote neovascularization. We evaluate the expression of netrin-1 during retinal neovascularization in a murine model of oxygen-induced retinopathy.
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
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pubmed:chemical |
|
pubmed:status |
MEDLINE
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pubmed:issn |
1423-0267
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pubmed:author |
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pubmed:copyrightInfo |
Copyright © 2011 S. Karger AG, Basel.
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pubmed:issnType |
Electronic
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pubmed:volume |
226
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pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
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pubmed:pagination |
37-44
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pubmed:meshHeading |
pubmed-meshheading:21508652-Animals,
pubmed-meshheading:21508652-Blood-Retinal Barrier,
pubmed-meshheading:21508652-Blotting, Western,
pubmed-meshheading:21508652-Capillary Permeability,
pubmed-meshheading:21508652-DNA Primers,
pubmed-meshheading:21508652-Dextrans,
pubmed-meshheading:21508652-Disease Models, Animal,
pubmed-meshheading:21508652-Fluorescein Angiography,
pubmed-meshheading:21508652-Fluoresceins,
pubmed-meshheading:21508652-Gene Expression,
pubmed-meshheading:21508652-Humans,
pubmed-meshheading:21508652-Infant, Newborn,
pubmed-meshheading:21508652-Mice,
pubmed-meshheading:21508652-Mice, Inbred C57BL,
pubmed-meshheading:21508652-Nerve Growth Factors,
pubmed-meshheading:21508652-Oxygen,
pubmed-meshheading:21508652-RNA, Messenger,
pubmed-meshheading:21508652-Retinal Neovascularization,
pubmed-meshheading:21508652-Retinal Vessels,
pubmed-meshheading:21508652-Retinopathy of Prematurity,
pubmed-meshheading:21508652-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:21508652-Tumor Suppressor Proteins
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pubmed:year |
2011
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pubmed:articleTitle |
Netrin-1 overexpression in oxygen-induced retinopathy correlates with breakdown of the blood-retina barrier and retinal neovascularization.
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pubmed:affiliation |
Department of Ophthalmology, Xiangya Hospital, Central South University, Changsha, China.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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