| pubmed-article:2139697 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2139697 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:2139697 | lifeskim:mentions | umls-concept:C0022686 | lld:lifeskim |
| pubmed-article:2139697 | lifeskim:mentions | umls-concept:C0034819 | lld:lifeskim |
| pubmed-article:2139697 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:2139697 | lifeskim:mentions | umls-concept:C1510827 | lld:lifeskim |
| pubmed-article:2139697 | lifeskim:mentions | umls-concept:C1515655 | lld:lifeskim |
| pubmed-article:2139697 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:2139697 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:2139697 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:2139697 | pubmed:dateCreated | 1990-6-6 | lld:pubmed |
| pubmed-article:2139697 | pubmed:abstractText | The majority of human NK cells express low affinity IgG Fc receptors (CD16+), whereas a minor subset of NK cells lack Fc receptor expression (CD16-). In contrast to CD16+ NK cells that express only p75 IL-2 receptors, CD16- NK cells constitutively co-express both p75 and p55 IL-2 receptors in vivo and preferentially respond to low concentrations of IL-2 with increased cytolytic activation and proliferation. Scatchard analysis demonstrated the presence of approximately 1,200 high affinity (approximately 25 pM kD) and approximately 9,600 intermediate affinity (approximately 2 nM kD) IL-2 receptors on CD16- NK cells. CD16+ NK cells expressed only a single intermediate affinity IL-2 receptor of approximately 1.9 nM kD (approximately 9,000 sites per cell). The IL-2 binding data thus substantiated the phenotypic and functional studies and definitively show that the differential responsiveness of CD16- and CD16+ NK cells to IL-2 is manifested through different affinity IL-2 receptors. | lld:pubmed |
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| pubmed-article:2139697 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2139697 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2139697 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2139697 | pubmed:month | May | lld:pubmed |
| pubmed-article:2139697 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:2139697 | pubmed:author | pubmed-author:PhillipsJ HJH | lld:pubmed |
| pubmed-article:2139697 | pubmed:author | pubmed-author:LanierL LLL | lld:pubmed |
| pubmed-article:2139697 | pubmed:author | pubmed-author:NaglerAA | lld:pubmed |
| pubmed-article:2139697 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2139697 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:2139697 | pubmed:volume | 171 | lld:pubmed |
| pubmed-article:2139697 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2139697 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2139697 | pubmed:pagination | 1527-33 | lld:pubmed |
| pubmed-article:2139697 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2139697 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2139697 | pubmed:articleTitle | Constitutive expression of high affinity interleukin 2 receptors on human CD16-natural killer cells in vivo. | lld:pubmed |
| pubmed-article:2139697 | pubmed:affiliation | Becton Dickinson Monoclonal Center, Inc., San Jose, California 95131. | lld:pubmed |
| pubmed-article:2139697 | pubmed:publicationType | Journal Article | lld:pubmed |
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