pubmed-article:21364961 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21364961 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:21364961 | lifeskim:mentions | umls-concept:C0553695 | lld:lifeskim |
pubmed-article:21364961 | lifeskim:mentions | umls-concept:C0034963 | lld:lifeskim |
pubmed-article:21364961 | lifeskim:mentions | umls-concept:C0205421 | lld:lifeskim |
pubmed-article:21364961 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:21364961 | pubmed:dateCreated | 2011-3-2 | lld:pubmed |
pubmed-article:21364961 | pubmed:abstractText | Telomere shortening is a cell-intrinsic mechanism that limits cell proliferation by induction of DNA damage responses resulting either in apoptosis or cellular senescence. Shortening of telomeres has been shown to occur during human aging and in chronic diseases that accelerate cell turnover, such as chronic hepatitis. Telomere shortening can limit organ homeostasis and regeneration in response to injury. Whether the same holds true for pancreas regeneration in response to injury is not known. | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:language | eng | lld:pubmed |
pubmed-article:21364961 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:21364961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21364961 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21364961 | pubmed:issn | 1932-6203 | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:AdlerGuidoG | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:RudolphKarl... | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:WagnerMartinM | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:von... | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:KlegerAlexand... | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:HartmannDanie... | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:NalapareddyKo... | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:GuachallaLuis... | lld:pubmed |
pubmed-article:21364961 | pubmed:author | pubmed-author:RolyanHarshva... | lld:pubmed |
pubmed-article:21364961 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:21364961 | pubmed:volume | 6 | lld:pubmed |
pubmed-article:21364961 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21364961 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21364961 | pubmed:pagination | e17122 | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:meshHeading | pubmed-meshheading:21364961... | lld:pubmed |
pubmed-article:21364961 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21364961 | pubmed:articleTitle | Regeneration of the exocrine pancreas is delayed in telomere-dysfunctional mice. | lld:pubmed |
pubmed-article:21364961 | pubmed:affiliation | Institute of Molecular Medicine and Max-Planck-Research-Group on Stem Cell Aging, University of Ulm, Ulm, Germany. | lld:pubmed |
pubmed-article:21364961 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:21364961 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
entrez-gene:21748 | entrezgene:pubmed | pubmed-article:21364961 | lld:entrezgene |
entrez-gene:21752 | entrezgene:pubmed | pubmed-article:21364961 | lld:entrezgene |
http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:21364961 | lld:entrezgene |
http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:21364961 | lld:entrezgene |