Inappropriate fetal exposure to maternal glucocorticoid (GC) has been proposed as a mechanism for fetal programming where the effects of GC may be mediated by the placenta. However, the consequences of maternal GC on placental morphology and enzyme expression are unclear.
Differential appearance of placentomes and expression of prostaglandin H synthase type 2 in placentome subtypes after betamethasone treatment of sheep late in gestation.
Department of Physiology and Obstetrics and Gynecology, University of Toronto, MSB, Rm. 3368, 1 King's College Circle, Toronto, Ontario M5S 1A8, Canada. thorsten.braun@charite.de
