rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2011-3-15
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pubmed:abstractText |
ARAP1 is an Arf (ADP-ribosylation factor)-directed GAP (GTPase-activating protein) that inhibits the trafficking of EGFR (epidermal growth factor receptor) to the early endosome. To further understand the function of ARAP1, we sought to identify proteins that interact with ARAP1.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/ARAP1 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/Adaptor Proteins, Signal Transducing,
http://linkedlifedata.com/resource/pubmed/chemical/Carrier Proteins,
http://linkedlifedata.com/resource/pubmed/chemical/Epidermal Growth Factor,
http://linkedlifedata.com/resource/pubmed/chemical/GTPase-Activating Proteins,
http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Epidermal Growth Factor,
http://linkedlifedata.com/resource/pubmed/chemical/SH3KBP1 protein, human
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
1768-322X
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pubmed:author |
|
pubmed:issnType |
Electronic
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pubmed:volume |
103
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
171-84
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pubmed:dateRevised |
2011-8-24
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pubmed:meshHeading |
pubmed-meshheading:21275903-Adaptor Proteins, Signal Transducing,
pubmed-meshheading:21275903-Carrier Proteins,
pubmed-meshheading:21275903-Endocytosis,
pubmed-meshheading:21275903-Epidermal Growth Factor,
pubmed-meshheading:21275903-GTPase-Activating Proteins,
pubmed-meshheading:21275903-HeLa Cells,
pubmed-meshheading:21275903-Humans,
pubmed-meshheading:21275903-Molecular Sequence Data,
pubmed-meshheading:21275903-Protein Binding,
pubmed-meshheading:21275903-Protein Transport,
pubmed-meshheading:21275903-Receptor, Epidermal Growth Factor
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pubmed:year |
2011
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pubmed:articleTitle |
ARAP1 association with CIN85 affects epidermal growth factor receptor endocytic trafficking.
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pubmed:affiliation |
Laboratory of Cellular and Molecular Biology, National Cancer Institute, Bethesda, MD 20892, U.S.A.
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pubmed:publicationType |
Journal Article,
Research Support, N.I.H., Extramural
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