rdf:type |
|
lifeskim:mentions |
umls-concept:C0012655,
umls-concept:C0013604,
umls-concept:C0014442,
umls-concept:C0030012,
umls-concept:C0031809,
umls-concept:C0162429,
umls-concept:C0205419,
umls-concept:C0314603,
umls-concept:C0439165,
umls-concept:C0439611,
umls-concept:C0596402,
umls-concept:C1155312,
umls-concept:C1280500,
umls-concept:C1533691
|
pubmed:issue |
1
|
pubmed:dateCreated |
2011-1-25
|
pubmed:abstractText |
The causes of oedematous vs non-oedematous childhood malnutrition (OM vs NOM) remain elusive. It is possible that inherited differences in handling oxidant stressors are a contributing factor.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:status |
MEDLINE
|
pubmed:issn |
1465-3281
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:volume |
31
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
27-36
|
pubmed:meshHeading |
pubmed-meshheading:21262107-Case-Control Studies,
pubmed-meshheading:21262107-Child,
pubmed-meshheading:21262107-Child, Preschool,
pubmed-meshheading:21262107-Child Nutrition Disorders,
pubmed-meshheading:21262107-Edema,
pubmed-meshheading:21262107-Female,
pubmed-meshheading:21262107-Genetic Predisposition to Disease,
pubmed-meshheading:21262107-Genotype,
pubmed-meshheading:21262107-Humans,
pubmed-meshheading:21262107-Infant,
pubmed-meshheading:21262107-Leukocytes, Mononuclear,
pubmed-meshheading:21262107-Oxidative Stress,
pubmed-meshheading:21262107-Pregnancy
|
pubmed:year |
2011
|
pubmed:articleTitle |
A preliminary examination of the effects of genetic variants of redox enzymes on susceptibility to oedematous malnutrition and on percentage cytotoxicity in response to oxidative stress in vitro.
|
pubmed:affiliation |
Tropical Metabolism Research Unit, University of the West Indies, Mona, Jamaica.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|