Linked Life Data

21235533

Source:http://linkedlifedata.com/resource/pubmed/id/21235533

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pubmed-article:21235533pubmed:abstractTextWe have evaluated the effects of the carbon monoxide-releasing molecule CORM-A1 [Na(2) (BH(3) CO(2) ); ALF421] on the development of relapsing-remitting experimental allergic encephalomyelitis (EAE) in SJL mice, an established model of multiple sclerosis (MS). The data show that the prolonged prophylactic administration of CORM-A1 improves the clinical and histopathological signs of EAE, as shown by a reduced cumulative score, shorter duration and a lower cumulative incidence of the disease as well as milder inflammatory infiltrations of the spinal cords. This study suggests that the use of CORM-A1 might represent a novel therapeutic strategy for the treatment of multiple sclerosis.lld:pubmed
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pubmed-article:21235533pubmed:copyrightInfo© 2011 The Authors. Clinical and Experimental Immunology © 2011 British Society for Immunology.lld:pubmed
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pubmed-article:21235533pubmed:dateRevised2011-8-26lld:pubmed
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pubmed-article:21235533pubmed:articleTitlePrevention of clinical and histological signs of proteolipid protein (PLP)-induced experimental allergic encephalomyelitis (EAE) in mice by the water-soluble carbon monoxide-releasing molecule (CORM)-A1.lld:pubmed
pubmed-article:21235533pubmed:affiliationDepartment of Biomedical Sciences, School of Medicine, University of Catania, Italy.lld:pubmed
pubmed-article:21235533pubmed:publicationTypeJournal Articlelld:pubmed