21177121

Source:http://linkedlifedata.com/resource/pubmed/id/21177121

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0003591, umls-concept:C0026809, umls-concept:C0031437, umls-concept:C0033414, umls-concept:C0332461, umls-concept:C1424460, umls-concept:C1514559
pubmed:issue	3
pubmed:dateCreated	2011-2-4
pubmed:abstractText	CXCL16 has been shown to be involved in atherosclerotic lesion development, but its role in preexisting lesions is still unclear. This study aims to assess the effect of CXCL16 on the stability of preexisting lesions.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9005353
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Apolipoproteins E, http://linkedlifedata.com/resource/pubmed/chemical/Ccl2 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CCL2, http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CXCL6, http://linkedlifedata.com/resource/pubmed/chemical/Cholesterol, Dietary, http://linkedlifedata.com/resource/pubmed/chemical/Cxcl16 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Matrix Metalloproteinases, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Necrosis Factor-alpha, http://linkedlifedata.com/resource/pubmed/chemical/Vascular Cell Adhesion Molecule-1
pubmed:status	MEDLINE
pubmed:month	Mar
pubmed:issn	1096-0023
pubmed:author	pubmed-author:ChengMinM, pubmed-author:JiQin-weiQW, pubmed-author:LiuHong-taoHT, pubmed-author:MaoXiao-boXB, pubmed-author:MaoYiY, pubmed-author:MoeJ FJF, pubmed-author:YangXiao-fangXF, pubmed-author:YiGui-wenGW, pubmed-author:ZengQiu-tangQT, pubmed-author:ZhongYu-chengYC
pubmed:copyrightInfo	Copyright © 2010 Elsevier Ltd. All rights reserved.
pubmed:issnType	Electronic
pubmed:volume	53
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	320-6
pubmed:meshHeading	pubmed-meshheading:21177121-Animals, pubmed-meshheading:21177121-Apolipoproteins E, pubmed-meshheading:21177121-Braces, pubmed-meshheading:21177121-Carotid Arteries, pubmed-meshheading:21177121-Chemokine CCL2, pubmed-meshheading:21177121-Chemokine CXCL6, pubmed-meshheading:21177121-Cholesterol, Dietary, pubmed-meshheading:21177121-Enzyme-Linked Immunosorbent Assay, pubmed-meshheading:21177121-Gene Expression, pubmed-meshheading:21177121-Male, pubmed-meshheading:21177121-Matrix Metalloproteinases, pubmed-meshheading:21177121-Mice, pubmed-meshheading:21177121-Mice, Inbred C57BL, pubmed-meshheading:21177121-Mice, Knockout, pubmed-meshheading:21177121-Phenotype, pubmed-meshheading:21177121-Plaque, Atherosclerotic, pubmed-meshheading:21177121-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:21177121-Transfection, pubmed-meshheading:21177121-Tumor Necrosis Factor-alpha, pubmed-meshheading:21177121-Vascular Cell Adhesion Molecule-1
pubmed:year	2011
pubmed:articleTitle	Overexpression of CXCL16 promotes a vulnerable plaque phenotype in Apolipoprotein E-Knockout Mice.
pubmed:affiliation	Institute of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, People's Republic of China.
pubmed:publicationType	Journal Article