| pubmed-article:21155037 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21155037 | lifeskim:mentions | umls-concept:C0021289 | lld:lifeskim |
| pubmed-article:21155037 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:21155037 | lifeskim:mentions | umls-concept:C0079459 | lld:lifeskim |
| pubmed-article:21155037 | lifeskim:mentions | umls-concept:C0273115 | lld:lifeskim |
| pubmed-article:21155037 | lifeskim:mentions | umls-concept:C1155266 | lld:lifeskim |
| pubmed-article:21155037 | lifeskim:mentions | umls-concept:C0599946 | lld:lifeskim |
| pubmed-article:21155037 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:21155037 | pubmed:dateCreated | 2010-12-14 | lld:pubmed |
| pubmed-article:21155037 | pubmed:abstractText | Granulocyte colony stimulating factor (G-CSF) has been known to increase neutrophil production and have anti-inflammatory properties, but the effect of G-CSF on pulmonary system is in controversy. We investigated whether G-CSF treatment could attenuate hyperoxia-induced lung injury, and whether this protective effect is mediated by the down-modulation of inflammatory responses in a neonatal rat model. | lld:pubmed |
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| pubmed-article:21155037 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21155037 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21155037 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:21155037 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21155037 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:21155037 | pubmed:issn | 1976-2437 | lld:pubmed |
| pubmed-article:21155037 | pubmed:author | pubmed-author:ParkWon... | lld:pubmed |
| pubmed-article:21155037 | pubmed:author | pubmed-author:ChangYun... | lld:pubmed |
| pubmed-article:21155037 | pubmed:author | pubmed-author:SungDong... | lld:pubmed |
| pubmed-article:21155037 | pubmed:author | pubmed-author:JungYu JinYJ | lld:pubmed |
| pubmed-article:21155037 | pubmed:author | pubmed-author:KooSoo HyunSH | lld:pubmed |
| pubmed-article:21155037 | pubmed:author | pubmed-author:JeonGa WonGW | lld:pubmed |
| pubmed-article:21155037 | pubmed:author | pubmed-author:SinJong... | lld:pubmed |
| pubmed-article:21155037 | pubmed:author | pubmed-author:ChoiSeo... | lld:pubmed |
| pubmed-article:21155037 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21155037 | pubmed:volume | 52 | lld:pubmed |
| pubmed-article:21155037 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21155037 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21155037 | pubmed:pagination | 65-73 | lld:pubmed |
| pubmed-article:21155037 | pubmed:dateRevised | 2011-7-20 | lld:pubmed |
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| pubmed-article:21155037 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21155037 | pubmed:articleTitle | Granulocyte colony stimulating factor attenuates hyperoxia-induced lung injury by down-modulating inflammatory responses in neonatal rats. | lld:pubmed |
| pubmed-article:21155037 | pubmed:affiliation | Department of Pediatrics, Pusan Paik Hospital, Inje University College of Medicine, Busan, Korea. | lld:pubmed |
| pubmed-article:21155037 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21155037 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:25610 | entrezgene:pubmed | pubmed-article:21155037 | lld:entrezgene |
