Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:dateCreated
2010-12-14
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
0065-2598
pubmed:author
pubmed:issnType
Print
pubmed:volume
691
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
409-19
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:21153346-Amino Acid Substitution, pubmed-meshheading:21153346-Animals, pubmed-meshheading:21153346-Anti-Inflammatory Agents, pubmed-meshheading:21153346-Antibodies, Monoclonal, pubmed-meshheading:21153346-Biological Agents, pubmed-meshheading:21153346-Family, pubmed-meshheading:21153346-Female, pubmed-meshheading:21153346-Hereditary Autoinflammatory Diseases, pubmed-meshheading:21153346-Humans, pubmed-meshheading:21153346-Immunoglobulin G, pubmed-meshheading:21153346-Ireland, pubmed-meshheading:21153346-Male, pubmed-meshheading:21153346-Mice, pubmed-meshheading:21153346-Mutant Proteins, pubmed-meshheading:21153346-Mutation, pubmed-meshheading:21153346-Pedigree, pubmed-meshheading:21153346-Receptors, Tumor Necrosis Factor, pubmed-meshheading:21153346-Receptors, Tumor Necrosis Factor, Type I, pubmed-meshheading:21153346-Treatment Failure, pubmed-meshheading:21153346-Tumor Necrosis Factors
pubmed:year
2011
pubmed:articleTitle
Lessons from anti-TNF biologics: infliximab failure in a TRAPS family with the T50M mutation in TNFRSF1A.
pubmed:affiliation
Centre for Diabetes, Blizard Institute of Cell and Molecular Science, Barts and The London School of Medicine and Dentistry, Whitechapel, London E1 2AT, UK. b.nedjai@imperial.ac.uk
pubmed:publicationType
Journal Article