pubmed-article:21123456 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21123456 | lifeskim:mentions | umls-concept:C1140680 | lld:lifeskim |
pubmed-article:21123456 | lifeskim:mentions | umls-concept:C0678133 | lld:lifeskim |
pubmed-article:21123456 | lifeskim:mentions | umls-concept:C0110116 | lld:lifeskim |
pubmed-article:21123456 | lifeskim:mentions | umls-concept:C0334227 | lld:lifeskim |
pubmed-article:21123456 | lifeskim:mentions | umls-concept:C0079083 | lld:lifeskim |
pubmed-article:21123456 | lifeskim:mentions | umls-concept:C1413474 | lld:lifeskim |
pubmed-article:21123456 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:21123456 | pubmed:dateCreated | 2011-3-3 | lld:pubmed |
pubmed-article:21123456 | pubmed:abstractText | We have previously shown that CLDN4 (encoding claudin-4), a cell tight junction (TJ) protein, is highly expressed in human epithelial ovarian carcinomas (EOC) but undetectable in normal ovaries. CLDN4 has been identified as a specific receptor for C terminus of Clostridium perfringens enterotoxin (C-CPE), a nontoxic molecule that may disrupt TJ barrier function and enhance cellular absorption. The purpose of this study was to determine the potential clinical applications of C-CPE and its effects on CLDN4 expression in EOC. | lld:pubmed |
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pubmed-article:21123456 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:language | eng | lld:pubmed |
pubmed-article:21123456 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:21123456 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:21123456 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21123456 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21123456 | pubmed:month | Mar | lld:pubmed |
pubmed-article:21123456 | pubmed:issn | 1078-0432 | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:MokSamuel CSC | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:BerkowitzRoss... | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:ZengQingQ | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:GarnerElizabe... | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:GaoZhijianZ | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:XuXiaoyinX | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:WelchWilliam... | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:McClaneBruceB | lld:pubmed |
pubmed-article:21123456 | pubmed:author | pubmed-author:LitkouhiBabak... | lld:pubmed |
pubmed-article:21123456 | pubmed:copyrightInfo | ©2010 AACR. | lld:pubmed |
pubmed-article:21123456 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:21123456 | pubmed:day | 1 | lld:pubmed |
pubmed-article:21123456 | pubmed:volume | 17 | lld:pubmed |
pubmed-article:21123456 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21123456 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21123456 | pubmed:pagination | 1065-74 | lld:pubmed |
pubmed-article:21123456 | pubmed:dateRevised | 2011-9-26 | lld:pubmed |
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pubmed-article:21123456 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21123456 | pubmed:articleTitle | C terminus of Clostridium perfringens enterotoxin downregulates CLDN4 and sensitizes ovarian cancer cells to Taxol and Carboplatin. | lld:pubmed |
pubmed-article:21123456 | pubmed:affiliation | Division of Gynecologic Oncology, Department of Obstetrics, Gynecology and Reproductive Biology, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts, USA. zhijian_gao@yahoo.com | lld:pubmed |
pubmed-article:21123456 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:21123456 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:21123456 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:21123456 | lld:pubmed |