| pubmed-article:21110863 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21110863 | lifeskim:mentions | umls-concept:C0026473 | lld:lifeskim |
| pubmed-article:21110863 | lifeskim:mentions | umls-concept:C0024432 | lld:lifeskim |
| pubmed-article:21110863 | lifeskim:mentions | umls-concept:C0268647 | lld:lifeskim |
| pubmed-article:21110863 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
| pubmed-article:21110863 | pubmed:dateCreated | 2010-12-14 | lld:pubmed |
| pubmed-article:21110863 | pubmed:abstractText | In the recessive aminoaciduria Lysinuric Protein Intolerance (LPI), mutations of SLC7A7/y+LAT1 impair system y+L transport activity for cationic amino acids. A severe complication of LPI is a form of Pulmonary Alveolar Proteinosis (PAP), in which alveolar spaces are filled with lipoproteinaceous material because of the impaired surfactant clearance by resident macrophages. The pathogenesis of LPI-associated PAP remains still obscure. The present study investigates for the first time the expression and function of y+LAT1 in monocytes and macrophages isolated from a patient affected by LPI-associated PAP. A comparison with mesenchymal cells from the same subject has been also performed. | lld:pubmed |
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| pubmed-article:21110863 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21110863 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21110863 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21110863 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:21110863 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21110863 | pubmed:issn | 1750-1172 | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:RotoliBianca... | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:VisigalliRoss... | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:BussolatiOvid... | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:GazzolaGian... | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:Dall'AstaVale... | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:LuisettiMauri... | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:BarilliAmelia... | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:MarianiFrance... | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:RodiGiuseppeG | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:KadijaZamirZ | lld:pubmed |
| pubmed-article:21110863 | pubmed:author | pubmed-author:RuzzaMaria... | lld:pubmed |
| pubmed-article:21110863 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21110863 | pubmed:volume | 5 | lld:pubmed |
| pubmed-article:21110863 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21110863 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21110863 | pubmed:pagination | 32 | lld:pubmed |
| pubmed-article:21110863 | pubmed:dateRevised | 2011-7-20 | lld:pubmed |
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| pubmed-article:21110863 | pubmed:meshHeading | pubmed-meshheading:21110863... | lld:pubmed |
| pubmed-article:21110863 | pubmed:meshHeading | pubmed-meshheading:21110863... | lld:pubmed |
| pubmed-article:21110863 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:21110863 | pubmed:articleTitle | In Lysinuric Protein Intolerance system y+L activity is defective in monocytes and in GM-CSF-differentiated macrophages. | lld:pubmed |
| pubmed-article:21110863 | pubmed:affiliation | Dipartimento di Medicina Sperimentale, Sezione di Patologia Generale e Clinica, Università degli Studi di Parma, Parma, Italy. | lld:pubmed |
| pubmed-article:21110863 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21110863 | pubmed:publicationType | Case Reports | lld:pubmed |
| pubmed-article:21110863 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
