21088756

Source:http://linkedlifedata.com/resource/pubmed/id/21088756

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0023745, umls-concept:C0026336, umls-concept:C0034693, umls-concept:C0034721, umls-concept:C0175677, umls-concept:C0205179, umls-concept:C0205314, umls-concept:C0336867, umls-concept:C0598528, umls-concept:C0679622, umls-concept:C0741968, umls-concept:C1514485, umls-concept:C2697979, umls-concept:C2828360
pubmed:issue	10
pubmed:dateCreated	2010-11-22
pubmed:abstractText	Vascular perturbation induced by advanced glycation end-products (AGEs) leads to progression of atherosclerosis, plaque instability, and vascular inflammation, which results in a higher risk of neointimal proliferation. Here we investigated the inhibitory effect of alagebrium chloride (ALT-711), a breaker of AGE-based cross links, on neointimal proliferation in a carotid artery balloon injury model in diabetic rats induced by streptozotocin (STZ).
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/101247141
pubmed:status	PubMed-not-MEDLINE
pubmed:month	Oct
pubmed:issn	1738-5555
pubmed:author	pubmed-author:ChaBong-SooBS, pubmed-author:HwangKi-ChulKC, pubmed-author:JangYangsooY, pubmed-author:KimJin-BaeJB, pubmed-author:LeeHyun-ChulHC, pubmed-author:LeeMoon-HyoungMH, pubmed-author:ParkSunghaS, pubmed-author:SongByeong-WookBW
pubmed:issnType	Electronic
pubmed:volume	40
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	520-6
pubmed:year	2010
pubmed:articleTitle	Alagebrium chloride, a novel advanced glycation end-product cross linkage breaker, inhibits neointimal proliferation in a diabetic rat carotid balloon injury model.
pubmed:affiliation	Cardiology Division, Yonsei University College of Medicine, Seoul, Korea.
pubmed:publicationType	Journal Article