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pubmed-article:21078800pubmed:abstractTextThe renin-angiotensin system (RAS) mediates proapoptotic, profibrotic, and proinflammatory processes in maladaptive conditions. Activation after myocardial infarction may initialize and promote cardiac remodeling. Using a novel positron-emitting ligand, we sought to determine the presence and time course of regional myocardial upregulation of the angiotensin II type 1 receptor (AT1R) and the blocking efficacy of various anti-RAS agents.lld:pubmed
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pubmed-article:21078800pubmed:articleTitleRadionuclide imaging of angiotensin II type 1 receptor upregulation after myocardial ischemia-reperfusion injury.lld:pubmed
pubmed-article:21078800pubmed:affiliationDivision of Nuclear Medicine, Russell H Morgan Department of Radiology and Radiological Sciences, Johns Hopkins University, Baltimore, Maryland, USA.lld:pubmed
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