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rdf:type |
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|
lifeskim:mentions |
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pubmed:issue |
11
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pubmed:dateCreated |
2010-11-30
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pubmed:abstractText |
Chitosan induced stomatal closure in wild type-plants and NADPH oxidase knock-out mutants (atrbohD atrbohF), and reactive oxygen species (ROS) production in wild-type guard cells. Closure and production were completely abolished by catalase and a peroxidase inhibitor. These results indicate that chitosan induces ROS production mediated by peroxidase, resulting in stomatal closure.
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pubmed:language |
eng
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pubmed:journal |
|
|
pubmed:citationSubset |
IM
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pubmed:chemical |
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|
pubmed:status |
MEDLINE
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pubmed:issn |
1347-6947
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pubmed:author |
|
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pubmed:issnType |
Electronic
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pubmed:volume |
74
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pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
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pubmed:pagination |
2313-5
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pubmed:dateRevised |
2011-8-11
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pubmed:meshHeading |
|
|
pubmed:year |
2010
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pubmed:articleTitle |
Chitosan-induced stomatal closure accompanied by peroxidase-mediated reactive oxygen species production in Arabidopsis.
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|
pubmed:affiliation |
Graduate School of Natural Science and Technology, Division of Bioscience, Okayama University, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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