Source:http://linkedlifedata.com/resource/pubmed/id/21047177
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
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pubmed:dateCreated |
2011-1-4
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pubmed:abstractText |
Cerebral ischemia triggers acute inflammation, which exacerbates primary brain damage. Characterization of cytokine expression to the early damaged tissue might aid in further understanding of lesion development and contribute to definition of molecular targets for selective immunotherapy. Endothelial monocyte-activating polypeptide II (EMAPII) is a proinflammatory, antiangiogenic cytokine whose expression following cerebral ischemia remained unknown. Therefore, we studied the spatiotemporal expression of EMAPII in early brain lesions after cerebral ischemia-reperfusion injury.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
1563-5279
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:volume |
121
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
58-64
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pubmed:meshHeading |
pubmed-meshheading:21047177-Animals,
pubmed-meshheading:21047177-Brain,
pubmed-meshheading:21047177-Brain Ischemia,
pubmed-meshheading:21047177-Cytokines,
pubmed-meshheading:21047177-Infarction, Middle Cerebral Artery,
pubmed-meshheading:21047177-Macrophages,
pubmed-meshheading:21047177-Male,
pubmed-meshheading:21047177-Microglia,
pubmed-meshheading:21047177-Neoplasm Proteins,
pubmed-meshheading:21047177-RNA-Binding Proteins,
pubmed-meshheading:21047177-Rats,
pubmed-meshheading:21047177-Rats, Sprague-Dawley,
pubmed-meshheading:21047177-Reperfusion Injury
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pubmed:year |
2011
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pubmed:articleTitle |
Lesional expression of EMAPII in macrophages/microglia following cerebral ischemia in rats.
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pubmed:affiliation |
Department of Traumatic Surgery, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, People's Republic of China.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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