pubmed-article:20961225 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20961225 | lifeskim:mentions | umls-concept:C0023861 | lld:lifeskim |
pubmed-article:20961225 | lifeskim:mentions | umls-concept:C0034790 | lld:lifeskim |
pubmed-article:20961225 | lifeskim:mentions | umls-concept:C0040549 | lld:lifeskim |
pubmed-article:20961225 | lifeskim:mentions | umls-concept:C0241526 | lld:lifeskim |
pubmed-article:20961225 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:20961225 | lifeskim:mentions | umls-concept:C0065082 | lld:lifeskim |
pubmed-article:20961225 | pubmed:issue | 11 | lld:pubmed |
pubmed-article:20961225 | pubmed:dateCreated | 2010-11-4 | lld:pubmed |
pubmed-article:20961225 | pubmed:abstractText | The success of many pathogens relies on their ability to circumvent the innate and adaptive immune defenses. How bacterial pathogens subvert adaptive immune defenses is not clear. Cholesterol-dependent cytolysins (CDCs) represent an expansive family of homologous pore-forming toxins that are produced by more than 20 gram-positive bacterial species. Listeriolysin O (LLO), a prototype CDC, is the main virulence factor of Listeria monocytogenes. | lld:pubmed |
pubmed-article:20961225 | pubmed:language | eng | lld:pubmed |
pubmed-article:20961225 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20961225 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:20961225 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20961225 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20961225 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20961225 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20961225 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20961225 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20961225 | pubmed:month | Dec | lld:pubmed |
pubmed-article:20961225 | pubmed:issn | 1537-6613 | lld:pubmed |
pubmed-article:20961225 | pubmed:author | pubmed-author:WeissSiegfrie... | lld:pubmed |
pubmed-article:20961225 | pubmed:author | pubmed-author:GeffersRobert... | lld:pubmed |
pubmed-article:20961225 | pubmed:author | pubmed-author:GekaraNelson... | lld:pubmed |
pubmed-article:20961225 | pubmed:author | pubmed-author:ZietaraNatali... | lld:pubmed |
pubmed-article:20961225 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20961225 | pubmed:day | 1 | lld:pubmed |
pubmed-article:20961225 | pubmed:volume | 202 | lld:pubmed |
pubmed-article:20961225 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20961225 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20961225 | pubmed:pagination | 1698-707 | lld:pubmed |
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pubmed-article:20961225 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20961225 | pubmed:articleTitle | Listeria monocytogenes induces T cell receptor unresponsiveness through pore-forming toxin listeriolysin O. | lld:pubmed |
pubmed-article:20961225 | pubmed:affiliation | Molecular Immunology, Helmholtz Center for Infection Research, Braunschweig, Germany. nelson.gekara@mims.umu.se | lld:pubmed |
pubmed-article:20961225 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20961225 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |