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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
2010-10-4
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pubmed:abstractText |
Thrombosis develops when the hemostatic system is incorrectly activated due to the unbalance between procoagulant, anticoagulant and fibrinolytic mechanisms allowing the formation of a clot within a blood vessel. The risk factors of this pathology can be acquired or can be genetic.
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pubmed:language |
spa
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
0120-4157
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
30
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
259-67
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pubmed:dateRevised |
2011-8-16
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pubmed:meshHeading |
pubmed-meshheading:20890573-Case-Control Studies,
pubmed-meshheading:20890573-Colombia,
pubmed-meshheading:20890573-Cystathionine beta-Synthase,
pubmed-meshheading:20890573-Female,
pubmed-meshheading:20890573-Homocysteine,
pubmed-meshheading:20890573-Humans,
pubmed-meshheading:20890573-Male,
pubmed-meshheading:20890573-Methylenetetrahydrofolate Reductase (NADPH2),
pubmed-meshheading:20890573-Middle Aged,
pubmed-meshheading:20890573-Polymorphism, Genetic,
pubmed-meshheading:20890573-Venous Thrombosis
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pubmed:articleTitle |
[Homocysteine levels and polymorphisms of MTHFR and CBS genes in Colombian patients with superficial and deep venous thrombosis].
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pubmed:affiliation |
Instituto de Genética Humana, Pontificia Universidad Javeriana, Bogotá, DC, Colombia.
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pubmed:publicationType |
Journal Article,
English Abstract,
Research Support, Non-U.S. Gov't
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